标题
Genetic deletion of CD36 enhances injury after acute neonatal stroke
作者
关键词
-
出版物
ANNALS OF NEUROLOGY
Volume 72, Issue 6, Pages 961-970
出版商
Wiley
发表日期
2012-08-10
DOI
10.1002/ana.23727
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Blood-Brain Barrier Permeability Is Increased After Acute Adult Stroke But Not Neonatal Stroke in the Rat
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- Microglial Cells Contribute to Endogenous Brain Defenses after Acute Neonatal Focal Stroke
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- Scavenger receptor CD36 is essential for the cerebrovascular oxidative stress and neurovascular dysfunction induced by amyloid-
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- Key Role of CD36 in Toll-Like Receptor 2 Signaling in Cerebral Ischemia
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- The Human Scavenger Receptor CD36
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- Differential trafficking of Src, Lyn, Yes and Fyn is specified by the state of palmitoylation in the SH4 domain
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- CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer
- (2009) Cameron R Stewart et al. NATURE IMMUNOLOGY
- CD36/Fatty Acid Translocase, An Inflammatory Mediator, Is Involved in Hyperlipidemia-Induced Exacerbation in Ischemic Brain Injury
- (2008) E. Kim et al. JOURNAL OF NEUROSCIENCE
- Nuclear Factor- B Activation and Postischemic Inflammation Are Suppressed in CD36-Null Mice after Middle Cerebral Artery Occlusion
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- Pharmacological and genetic inhibition of NADPH oxidase does not reduce brain damage in different models of perinatal brain injury in newborn mice
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- Strong Neuroprotection by Inhibition of NF-κB After Neonatal Hypoxia-Ischemia Involves Apoptotic Mechanisms but Is Independent of Cytokines
- (2008) Cora H.A. Nijboer et al. STROKE
- Hematoma Resolution as a Therapeutic Target: The Role of Microglia/Macrophages
- (2008) X. Zhao et al. STROKE
- A Dual Role of the NF-κB Pathway in Neonatal Hypoxic-Ischemic Brain Damage
- (2008) Cora H. Nijboer et al. STROKE
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