Reciprocal Th1 and Th17 regulation by mesenchymal stem cells: Implication for multiple sclerosis
出版年份 2010 全文链接
标题
Reciprocal Th1 and Th17 regulation by mesenchymal stem cells: Implication for multiple sclerosis
作者
关键词
-
出版物
ANNALS OF NEUROLOGY
Volume 68, Issue 4, Pages 540-545
出版商
Wiley
发表日期
2010-07-26
DOI
10.1002/ana.22065
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- The therapeutic potential of mesenchymal stem cell transplantation as a treatment for multiple sclerosis: consensus report of the International MSCT Study Group
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- Multipotent mesenchymal stromal cells for autoimmune diseases: teaching new dogs old tricks
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- Fetal BM-derived mesenchymal stem cells promote the expansion of human Th17 cells, but inhibit the production of Th1 cells
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- Human bone marrow-derived mesenchymal stem cells induce Th2-polarized immune response and promote endogenous repair in animal models of multiple sclerosis
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- Mesenchymal stem cells efficiently inhibit the proinflammatory properties of 6-sulfo LacNAc dendritic cells
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- Mesenchymal Stromal Cells Ameliorate Experimental Autoimmune Encephalomyelitis by Inhibiting CD4 Th17 T Cells in a CC Chemokine Ligand 2-Dependent Manner
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- Mixed results with modulation of TH-17 cells in human autoimmune diseases
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- MicroRNA miR-326 regulates TH-17 differentiation and is associated with the pathogenesis of multiple sclerosis
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- Mesenchymal Stem Cell-Mediated Immunosuppression Occurs via Concerted Action of Chemokines and Nitric Oxide
- (2008) Guangwen Ren et al. Cell Stem Cell
- The potential use of stem cells in multiple sclerosis: An overview of the preclinical experience
- (2008) Dimitrios Karussis et al. CLINICAL NEUROLOGY AND NEUROSURGERY
- T helper cell type 1 (Th1), Th2 and Th17 responses to myelin basic protein and disease activity in multiple sclerosis
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- Differences in mesenchymal stem cell cytokine profiles between MS patients and healthy donors: Implication for assessment of disease activity and treatment
- (2008) Benedetta Mazzanti et al. JOURNAL OF NEUROIMMUNOLOGY
- Human mesenchymal stem cells abrogate experimental allergic encephalomyelitis after intraperitoneal injection, and with sparse CNS infiltration
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