Endogenous α2-Antiplasmin Is Protective during Severe Gram-Negative Sepsis (Melioidosis)

Endogenous tissue-type plasminogen activator impairs host defense during severe experimental gram-negative sepsis (melioidosis)*

(2012) Liesbeth M. Kager et al.   CRITICAL CARE MEDICINE

Expression and Function of Transforming Growth Factor β in Melioidosis

(2012) Tassili A. F. Weehuizen et al.   INFECTION AND IMMUNITY

Viral research faces clampdown

(2012) Declan Butler   NATURE

Melioidosis

(2012) W. Joost Wiersinga et al.   NEW ENGLAND JOURNAL OF MEDICINE

Plasminogen activator inhibitor type I contributes to protective immunity during experimental Gram-negative sepsis (melioidosis)

(2011) L. M. KAGER et al.   JOURNAL OF THROMBOSIS AND HAEMOSTASIS

Inflammation and coagulation

(2010) Marcel Levi et al.   CRITICAL CARE MEDICINE

α2-Antiplasmin and its deficiency: fibrinolysis out of balance

(2010) S. L. CARPENTER et al.   HAEMOPHILIA

Cellular and molecular basis for the regulation of inflammation by TGF- 

(2010) A. Yoshimura et al.   JOURNAL OF BIOCHEMISTRY

The Epidemiology and Clinical Spectrum of Melioidosis: 540 Cases from the 20 Year Darwin Prospective Study

(2010) Bart J. Currie et al.   PLoS Neglected Tropical Diseases

α2-Antiplasmin Is Associated with the Progression of Fibrosis

(2009) Yosuke Kanno et al.   AMERICAN JOURNAL OF PATHOLOGY