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Gsα deficiency in skeletal muscle leads to reduced muscle mass, fiber-type switching, and glucose intolerance without insulin resistance or deficiency

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Androgen Signaling in Myocytes Contributes to the Maintenance of Muscle Mass and Fiber Type Regulation But Not to Muscle Strength or Fatigue

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Regulation of ClC-1 and KATPchannels in action potential–firing fast-twitch muscle fibers

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Contractile dysfunctions in ATP-dependent K+channel-deficient mouse muscle during fatigue involve excessive depolarization and Ca2+influx through L-type Ca2+channels

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Muscle K+, Na+, and Cl− disturbances and Na+-K+ pump inactivation: implications for fatigue

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