Betulinic acid regulates generation of neuroinflammatory mediators responsible for tissue destruction in multiple sclerosis in vitro
出版年份 2013 全文链接
标题
Betulinic acid regulates generation of neuroinflammatory mediators responsible for tissue destruction in multiple sclerosis in vitro
作者
关键词
-
出版物
ACTA PHARMACOLOGICA SINICA
Volume 34, Issue 3, Pages 424-431
出版商
Springer Nature
发表日期
2013-02-04
DOI
10.1038/aps.2012.181
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- New and emerging disease modifying therapies for multiple sclerosis
- (2012) Shiv Saidha et al. Annals of the New York Academy of Sciences
- Effect of betulinic acid on neutrophil recruitment and inflammatory mediator expression in lipopolysaccharide-induced lung inflammation in rats
- (2012) Manar A. Nader et al. EUROPEAN JOURNAL OF PHARMACEUTICAL SCIENCES
- CXCL12 in control of neuroinflammation
- (2012) Miljana Momcilović et al. IMMUNOLOGIC RESEARCH
- Neuroinflammation: A Common Pathway in CNS Diseases as Mediated at the Blood-Brain Barrier
- (2012) Michelle A. Erickson et al. NEUROIMMUNOMODULATION
- Oxidative damage in multiple sclerosis lesions
- (2011) L. Haider et al. BRAIN
- Experimental autoimmune encephalomyelitis (EAE) as a model for multiple sclerosis (MS)
- (2011) Cris S Constantinescu et al. BRITISH JOURNAL OF PHARMACOLOGY
- Nanostructures for Drug Delivery to the Brain
- (2011) L. Martin-Banderas et al. CURRENT MEDICINAL CHEMISTRY
- Astrocytes in the tempest of multiple sclerosis
- (2011) Djordje Miljković et al. FEBS LETTERS
- Chronic inflammation and immunologic-based constraints in malignant disease
- (2011) Devin B Lowe et al. Immunotherapy
- Betulinic acid protects against cerebral ischemia–reperfusion injury in mice by reducing oxidative and nitrosative stress
- (2011) Qing Lu et al. NITRIC OXIDE-BIOLOGY AND CHEMISTRY
- Correction: Defining Natural History: Assessment of the Ability of College Students to Aid in Characterizing Clinical Progression of Niemann-Pick Disease, Type C
- (2011) Jenny Shin et al. PLoS One
- Triterpenoid modulation of IL-17 and Nrf-2 expression ameliorates neuroinflammation and promotes remyelination in autoimmune encephalomyelitis
- (2011) Tej K. Pareek et al. Scientific Reports
- Radical changes in multiple sclerosis pathogenesis
- (2010) Jack van Horssen et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE
- Carbamate derivatives of betulinic acid and betulin with selective cytotoxic activity
- (2010) Harish Kommera et al. BIOORGANIC & MEDICINAL CHEMISTRY LETTERS
- Small structural changes of pentacyclic lupane type triterpenoid derivatives lead to significant differences in their anticancer properties
- (2010) Harish Kommera et al. EUROPEAN JOURNAL OF MEDICINAL CHEMISTRY
- Current Views on the Roles of Th1 and Th17 Cells in Experimental Autoimmune Encephalomyelitis
- (2010) Mohamed El-behi et al. Journal of Neuroimmune Pharmacology
- Lupane Triterpenoids—Betulin and Betulinic acid derivatives induce apoptosis in tumor cells
- (2009) Harish Kommera et al. INVESTIGATIONAL NEW DRUGS
- Strain difference in susceptibility to experimental autoimmune encephalomyelitis in rats correlates with TH1 and TH17-inducing cytokine profiles
- (2009) Milos Markovic et al. MOLECULAR IMMUNOLOGY
- Kinetics of IFN-γ and IL-17 expression and production in active experimental autoimmune encephalomyelitis in Dark Agouti rats
- (2008) Miljana Momčilović et al. NEUROSCIENCE LETTERS
- Interleukin-17 Production in Central Nervous System-Infiltrating T Cells and Glial Cells Is Associated with Active Disease in Multiple Sclerosis
- (2007) John S. Tzartos et al. AMERICAN JOURNAL OF PATHOLOGY
- Review: Oxidative stress and excitotoxicity: a therapeutic issue in multiple sclerosis?
- (2007) R.E. Gonsette Multiple Sclerosis Journal
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started