标题
SERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse models
作者
关键词
-
出版物
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
Volume 308, Issue 9, Pages C699-C709
出版商
American Physiological Society
发表日期
2015-02-05
DOI
10.1152/ajpcell.00341.2014
参考文献
相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。- The role of proteases in excitation-contraction coupling failure in muscular dystrophy
- (2015) Davi A. G. Mázala et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Can't live with or without it: calcium and its role in Duchenne muscular dystrophy-induced muscle weakness. Focus on “SERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse models”
- (2015) Arthur J. Cheng et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Enhanced Ca2+ influx from STIM1–Orai1 induces muscle pathology in mouse models of muscular dystrophy
- (2014) Sanjeewa A. Goonasekera et al. HUMAN MOLECULAR GENETICS
- Myofiber branching rather than myofiber hyperplasia contributes to muscle hypertrophy in mdx mice
- (2014) Rachel M Faber et al. Skeletal Muscle
- Increased sarcolipin expression and decreased sarco(endo)plasmic reticulum Ca2+ uptake in skeletal muscles of mouse models of Duchenne muscular dystrophy
- (2013) Joel S. Schneider et al. JOURNAL OF MUSCLE RESEARCH AND CELL MOTILITY
- Effect of Calstabin1 Depletion on Calcium Transients and Energy Utilization in Muscle Fibers and Treatment Opportunities with RyR1 Stabilizers
- (2013) Anke Breckner et al. PLoS One
- Pathways of Ca2+ entry and cytoskeletal damage following eccentric contractions in mouse skeletal muscle
- (2012) Bao-Ting Zhang et al. JOURNAL OF APPLIED PHYSIOLOGY
- Ca2+-dependent proteolysis of junctophilin-1 and junctophilin-2 in skeletal and cardiac muscle
- (2012) R. M. Murphy et al. JOURNAL OF PHYSIOLOGY-LONDON
- Effects ofin vivoinjury on the neuromuscular junction in healthy and dystrophic muscles
- (2012) Stephen J.P. Pratt et al. JOURNAL OF PHYSIOLOGY-LONDON
- Hsp72 preserves muscle function and slows progression of severe muscular dystrophy
- (2012) Stefan M. Gehrig et al. NATURE
- Stretch-activated calcium channel protein TRPC1 is correlated with the different degrees of the dystrophic phenotype in mdx mice
- (2011) Cíntia Yuri Matsumura et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- An in vivo rodent model of contraction-induced injury in the quadriceps muscle
- (2011) Stephen J.P. Pratt et al. INJURY-INTERNATIONAL JOURNAL OF THE CARE OF THE INJURED
- TAT-μUtrophin mitigates the pathophysiology of dystrophin and utrophin double-knockout mice
- (2011) Jarrod A. Call et al. JOURNAL OF APPLIED PHYSIOLOGY
- Mitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle
- (2011) Sanjeewa A. Goonasekera et al. JOURNAL OF CLINICAL INVESTIGATION
- A single 30min treadmill exercise session is suitable for ‘proof-of concept studies’ in adult mdx mice: A comparison of the early consequences of two different treadmill protocols
- (2011) Hannah Radley-Crabb et al. NEUROMUSCULAR DISORDERS
- Upregulation of store-operated Ca2+ entry in dystrophic mdx mouse muscle
- (2010) Joshua N. Edwards et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Age-Related Differences in Lower-Limb Muscle Cross-Sectional Area and Torque Production in Boys With Duchenne Muscular Dystrophy
- (2010) Sunita Mathur et al. ARCHIVES OF PHYSICAL MEDICINE AND REHABILITATION
- Calcium and the damage pathways in muscular dystrophyThis article is one of a selection of papers published in this special issue on Calcium Signaling.
- (2010) David G. Allen et al. CANADIAN JOURNAL OF PHYSIOLOGY AND PHARMACOLOGY
- Overexpression of SERCA1a in the mdx Diaphragm Reduces Susceptibility to Contraction-Induced Damage
- (2010) Kevin J. Morine et al. HUMAN GENE THERAPY
- Genotyping mdx, mdx3cv, and mdx4cv mice by primer competition polymerase chain reaction
- (2010) Jin-Hong Shin et al. MUSCLE & NERVE
- Marginal Level Dystrophin Expression Improves Clinical Outcome in a Strain of Dystrophin/Utrophin Double Knockout Mice
- (2010) Dejia Li et al. PLoS One
- Malformed mdx myofibers have normal cytoskeletal architecture yet altered EC coupling and stress-induced Ca2+ signaling
- (2009) Richard M. Lovering et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Calpain-3 is activated following eccentric exercise
- (2009) Robyn M. Murphy et al. JOURNAL OF APPLIED PHYSIOLOGY
- Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanism
- (2009) Douglas P. Millay et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Stimulation of calcineurin Aα activity attenuates muscle pathophysiology in mdx dystrophic mice
- (2008) Nicole Stupka et al. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY
- Investigation of Debio 025, a cyclophilin inhibitor, in the dystrophic mdx mouse, a model for Duchenne muscular dystrophy
- (2008) J Reutenauer et al. BRITISH JOURNAL OF PHARMACOLOGY
- Effect of calpain and proteasome inhibition on Ca2+-dependent proteolysis and muscle histopathology in the mdx mouse
- (2008) Alexandre Briguet et al. FASEB JOURNAL
- Role of the calcium-calpain pathway in cytoskeletal damage after eccentric contractions
- (2008) Bao-Ting Zhang et al. JOURNAL OF APPLIED PHYSIOLOGY
- TRPC1 binds to caveolin-3 and is regulated by Src kinase - role in Duchenne muscular dystrophy
- (2008) O. L. Gervasio et al. JOURNAL OF CELL SCIENCE
- Systemic humanminidystrophingene transfer improves functions and life span of dystrophin and dystrophin/utrophin-deficient mice
- (2008) Bing Wang et al. JOURNAL OF ORTHOPAEDIC RESEARCH
- Towards developing standard operating procedures for pre-clinical testing in the mdx mouse model of Duchenne muscular dystrophy
- (2008) Miranda D. Grounds et al. NEUROBIOLOGY OF DISEASE
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More