标题
γδT Cells Exacerbate Podocyte Injury via the CD28/B7-1-Phosphor-SRC Kinase Pathway
作者
关键词
-
出版物
Biomed Research International
Volume 2018, Issue -, Pages 1-14
出版商
Hindawi Limited
发表日期
2018-05-16
DOI
10.1155/2018/5647120
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- IL-23 Activated γδ T Cells Affect Th17 Cells and Regulatory T Cells by Secreting IL-21 in Children with Primary Nephrotic Syndrome
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- γδ T Cells Support Pancreatic Oncogenesis by Restraining αβ T Cell Activation
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- Coinhibitory Pathways in the B7-CD28 Ligand-Receptor Family
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- Synaptopodin Is a Coincidence Detector of Tyrosine versus Serine/Threonine Phosphorylation for the Modulation of Rho Protein Crosstalk in Podocytes
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- A novel podocyte gene, semaphorin 3G, protects glomerular podocyte from lipopolysaccharide-induced inflammation
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- CTLA4-Ig in B7-1-positive diabetic and non-diabetic kidney disease
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- IL-1 receptor antagonist-deficient mice develop autoimmune arthritis due to intrinsic activation of IL-17-producing CCR2+Vγ6+γδ T cells
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- LPS Nephropathy in Mice Is Ameliorated by IL-2 Independently of Regulatory T Cells Activity
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- The origin and fate of γδT cell subsets
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- Abatacept in B7-1–Positive Proteinuric Kidney Disease
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- IL-17A Production by Renal T Cells Promotes Kidney Injury in Crescentic GN
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- IL-2/IL-2Ab Complexes Induce Regulatory T Cell Expansion and Protect against Proteinuric CKD
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- γδ T-cells: cross-talk between innate and adaptive immunity
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- Urinary CD80 is elevated in minimal change disease but not in focal segmental glomerulosclerosis
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- The prevalence of Th17 cells and FOXP3 regulate T cells (Treg) in children with primary nephrotic syndrome
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- Urinary CD80 Excretion Increases in Idiopathic Minimal-Change Disease
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