标题
TRIM proteins: New players in virus-induced autophagy
作者
关键词
Autophagic cell death, Viral pathogens, Viral packaging, Cargo proteins, Pattern recognition receptors, Interferons, Sindbis virus, Dengue virus
出版物
PLoS Pathogens
Volume 14, Issue 2, Pages e1006787
出版商
Public Library of Science (PLoS)
发表日期
2018-02-02
DOI
10.1371/journal.ppat.1006787
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- TRIM23 mediates virus-induced autophagy via activation of TBK1
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- TRIM5α Degradation via Autophagy Is Not Required for Retroviral Restriction
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- Receptor usage dictates HIV-1 restriction by human TRIM5α in dendritic cell subsets
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- Phosphorylation of OPTN by TBK1 enhances its binding to Ub chains and promotes selective autophagy of damaged mitochondria
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- TRIM Proteins Regulate Autophagy and Can Target Autophagic Substrates by Direct Recognition
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- Cargo recognition and trafficking in selective autophagy
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- Nonstructural proteins 2C and 3D are involved in autophagy as induced by the encephalomyocarditis virus
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- TBK-1 Promotes Autophagy-Mediated Antimicrobial Defense by Controlling Autophagosome Maturation
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- The Mitochondrial Proteins NLRX1 and TUFM Form a Complex that Regulates Type I Interferon and Autophagy
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