Down-regulation of connexin43 and connexin32 in keratocystic odontogenic tumours: potential association with clinical features
出版年份 2015 全文链接
标题
Down-regulation of connexin43 and connexin32 in keratocystic odontogenic tumours: potential association with clinical features
作者
关键词
-
出版物
HISTOPATHOLOGY
Volume 66, Issue 6, Pages 798-807
出版商
Wiley
发表日期
2014-10-01
DOI
10.1111/his.12569
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Downregulation of the transforming growth factor-β/connective tissue growth factor 2 signalling pathway in venous malformations: its target potential for sclerotherapy
- (2014) J.-G. Ren et al. BRITISH JOURNAL OF DERMATOLOGY
- Increased expression of autophagy-related proteins in keratocystic odontogenic tumours: its possible association with growth potential
- (2014) Rui-Fang Li et al. BRITISH JOURNAL OF ORAL & MAXILLOFACIAL SURGERY
- Connexins: Mechanisms regulating protein levels and intercellular communication
- (2014) Vivian Su et al. FEBS LETTERS
- Degradation of connexins and gap junctions
- (2014) Matthias M. Falk et al. FEBS LETTERS
- New insights into the role of connexins in pancreatic islet function and diabetes
- (2014) Nikki L. Farnsworth et al. FEBS LETTERS
- Connexins in colorectal cancer pathogenesis
- (2014) Solveig Sirnes et al. INTERNATIONAL JOURNAL OF CANCER
- Loss of connexin43 expression in Ewing's sarcoma cells favors the development of the primary tumor and the associated bone osteolysis
- (2013) Julie Talbot et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE
- Connexin a check-point component of cell apoptosis in normal and physiopathological conditions
- (2013) Diane Carette et al. BIOCHIMIE
- Bleomycin induces endothelial mesenchymal transition through activation of mTOR pathway: a possible mechanism contributing to the sclerotherapy of venous malformations
- (2013) Wei Zhang et al. BRITISH JOURNAL OF PHARMACOLOGY
- Hypoxia-induced autophagy in endothelial cells: a double-edged sword in the progression of infantile haemangioma?
- (2013) Gang Chen et al. CARDIOVASCULAR RESEARCH
- Internalized gap junctions are degraded by autophagy
- (2012) John T. Fong et al. Autophagy
- Degradation of Connexins Through the Proteasomal, Endolysosomal and Phagolysosomal Pathways
- (2012) Vivian Su et al. JOURNAL OF MEMBRANE BIOLOGY
- Autophagy modulates dynamics of connexins at the plasma membrane in a ubiquitin-dependent manner
- (2012) Eloy Bejarano et al. MOLECULAR BIOLOGY OF THE CELL
- The role of connexins in prostate cancer promotion and progression
- (2012) Jarosław Czyż et al. Nature Reviews Urology
- Autophagy: a pathway that contributes to connexin degradation
- (2011) A. Lichtenstein et al. JOURNAL OF CELL SCIENCE
- Image Analysis Tools for Evaluation of Microscopic Views of Immunohistochemically Stained Specimen in Medical Research–a Review
- (2011) Keerthana Prasad et al. JOURNAL OF MEDICAL SYSTEMS
- Cytoplasmic accumulation of connexin32 expands cancer stem cell population in human HuH7 hepatoma cells by enhancing its self-renewal
- (2010) Yohei Kawasaki et al. INTERNATIONAL JOURNAL OF CANCER
- Characterization and management of the keratocystic odontogenic tumor in relation to its histopathological and biological features
- (2010) Rui Amaral Mendes et al. ORAL ONCOLOGY
- Overexpression of Protease-Activated Receptor-1 Contributes to Melanoma Metastasis via Regulation of Connexin 43
- (2009) G. J. Villares et al. CANCER RESEARCH
- Gap Junction Turnover Is Achieved by the Internalization of Small Endocytic Double-Membrane Vesicles
- (2009) Matthias M. Falk et al. MOLECULAR BIOLOGY OF THE CELL
- Odontogenic tumors: A study of 1309 cases in a Chinese population
- (2009) Hai-Yan Luo et al. ORAL ONCOLOGY
- Gap Junctions and Cancer: New Functions for an Old Story
- (2008) Laurent Cronier et al. ANTIOXIDANTS & REDOX SIGNALING
Create your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create NowBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started