Protein interactions of FAM134B with EB1 and APC/beta-catenin in vitro in colon carcinoma
出版年份 2018 全文链接
标题
Protein interactions of FAM134B with EB1 and APC/beta-catenin in vitro in colon carcinoma
作者
关键词
-
出版物
MOLECULAR CARCINOGENESIS
Volume -, Issue -, Pages -
出版商
Wiley
发表日期
2018-07-02
DOI
10.1002/mc.22871
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- RETREG1 (FAM134B ): A new player in human diseases: 15 years after the discovery in cancer
- (2018) Farhadul Islam et al. JOURNAL OF CELLULAR PHYSIOLOGY
- Cribriform-morular variant of papillary thyroid carcinoma: a distinctive type of thyroid cancer
- (2017) Alfred King-yin Lam et al. ENDOCRINE-RELATED CANCER
- Cellular expression, in-vitro and in-vivo confirmation of GAEC1 oncogenic properties in colon cancer
- (2017) Riajul Wahab et al. EUROPEAN JOURNAL OF CELL BIOLOGY
- MicroRNA-186-5p overexpression modulates colon cancer growth by repressing the expression of the FAM134B tumour inhibitor
- (2017) Farhadul Islam et al. EXPERIMENTAL CELL RESEARCH
- Novel FAM134B mutations and their clinicopathological significance in colorectal cancer
- (2017) Farhadul Islam et al. HUMAN GENETICS
- EB1 protein alteration characterizes sporadic but not ulcerative colitis associated colorectal cancer
- (2017) Timo Gemoll et al. Oncotarget
- Fancd2 in vivo interaction network reveals a non-canonical role in mitochondrial function
- (2017) Tingting Zhang et al. Scientific Reports
- Integrated diagnostic network construction reveals a 4-gene panel and 5 cancer hallmarks driving breast cancer heterogeneity
- (2017) Xiaofeng Dai et al. Scientific Reports
- Targeting the interaction of Aurora kinases and SIRT1 mediated by Wnt signaling pathway in colorectal cancer: A critical review
- (2016) Boopathi Subramaniyan et al. BIOMEDICINE & PHARMACOTHERAPY
- FAM134B, the Selective Autophagy Receptor for Endoplasmic Reticulum Turnover, Inhibits Replication of Ebola Virus Strains Makona and Mayinga
- (2016) Abhilash I. Chiramel et al. JOURNAL OF INFECTIOUS DISEASES
- Stage dependent expression and tumor suppressive function ofFAM134B(JK1) in colon cancer
- (2016) Farhadul Islam et al. MOLECULAR CARCINOGENESIS
- Overexpression of CAP1 and its significance in tumor cell proliferation, migration and invasion in glioma
- (2016) Yue-Chao Fan et al. ONCOLOGY REPORTS
- Identification of Novel FAM134B (JK1) Mutations in Oesophageal Squamous Cell Carcinoma
- (2016) Md. Hakimul Haque et al. Scientific Reports
- Regulation of endoplasmic reticulum turnover by selective autophagy
- (2015) Aliaksandr Khaminets et al. NATURE
- Effects of gene silencing of CypB on gastric cancer cells
- (2015) Feng Guo et al. Asian Pacific Journal of Tropical Medicine
- A Golgi-based KDELR-dependent signalling pathway controls extracellular matrix degradation
- (2015) Carmen Ruggiero et al. Oncotarget
- JK1 (FAM134B) gene and colorectal cancer: A pilot study on the gene copy number alterations and correlations with clinicopathological parameters
- (2014) Kais Kasem et al. EXPERIMENTAL AND MOLECULAR PATHOLOGY
- JK1 (FAM134B) represses cell migration in colon cancer: a functional study of a novel gene
- (2014) Kais Kasem et al. EXPERIMENTAL AND MOLECULAR PATHOLOGY
- The roles of JK-1 (FAM134B) expressions in colorectal cancer
- (2014) Kais Kasem et al. EXPERIMENTAL CELL RESEARCH
- EB1 enables spindle microtubules to regulate centromeric recruitment of Aurora B
- (2014) Budhaditya Banerjee et al. JOURNAL OF CELL BIOLOGY
- Overexpression of adenylate cyclase-associated protein 1 may predict brain metastasis in non-small cell lung cancer
- (2014) SHUAN-SHUAN XIE et al. ONCOLOGY REPORTS
- Depletion of end-binding protein 1 (EB1) promotes apoptosis of human non-small-cell lung cancer cells via reactive oxygen species and Bax-mediated mitochondrial dysfunction
- (2013) Min-Jung Kim et al. CANCER LETTERS
- Mutation inFAM134Bcausing hereditary sensory neuropathy with spasticity in a Turkish family
- (2013) Elif Ilgaz Aydinlar et al. MUSCLE & NERVE
- Novel Targets in Non-Small Cell Lung Cancer: ROS1 and RET Fusions
- (2013) J. F. Gainor et al. ONCOLOGIST
- Frequency of mutations in the genes associated with hereditary sensory and autonomic neuropathy in a UK cohort
- (2012) G. L. Davidson et al. JOURNAL OF NEUROLOGY
- In-depth Proteomic Analysis of Nonsmall Cell Lung Cancer to Discover Molecular Targets and Candidate Biomarkers
- (2012) Takefumi Kikuchi et al. MOLECULAR & CELLULAR PROTEOMICS
- Clinicopathological significance of synchronous carcinoma in colorectal cancer
- (2011) Alfred King-Yin Lam et al. AMERICAN JOURNAL OF SURGERY
- Learning Biomarkers of Pluripotent Stem Cells in Mouse
- (2011) L. Scheubert et al. DNA RESEARCH
- Role of cyclophilin B in tumorigenesis and cisplatin resistance in hepatocellular carcinoma in humans
- (2011) Yeonghwan Kim et al. HEPATOLOGY
- Novel protein identification methods for biomarker discovery via a proteomic analysis of periodontally healthy and diseased gingival crevicular fluid samples
- (2011) Richard C. Baliban et al. JOURNAL OF CLINICAL PERIODONTOLOGY
- Mutation in FAM134B causing severe hereditary sensory neuropathy: Figure 1
- (2010) Sinéad M Murphy et al. JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY
- A strong synergistic epistasis between FAM134B and TNFRSF19 on the susceptibility to vascular dementia
- (2010) Minyoung Kong et al. PSYCHIATRIC GENETICS
- Mutations in FAM134B, encoding a newly identified Golgi protein, cause severe sensory and autonomic neuropathy
- (2009) Ingo Kurth et al. NATURE GENETICS
- Expression of Cyclophilin B is Associated with Malignant Progression and Regulation of Genes Implicated in the Pathogenesis of Breast Cancer
- (2008) Feng Fang et al. AMERICAN JOURNAL OF PATHOLOGY
- Aurora kinase expression in colorectal adenocarcinoma: correlations with clinicopathological features, p16 expression, and telomerase activity
- (2008) Alfred King-Yin Lam et al. HUMAN PATHOLOGY
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationCreate your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create Now