The AHR represses nucleotide excision repair and apoptosis and contributes to UV-induced skin carcinogenesis
出版年份 2018 全文链接
标题
The AHR represses nucleotide excision repair and apoptosis and contributes to UV-induced skin carcinogenesis
作者
关键词
-
出版物
CELL DEATH AND DIFFERENTIATION
Volume -, Issue -, Pages -
出版商
Springer Nature
发表日期
2018-07-16
DOI
10.1038/s41418-018-0160-1
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Aryl hydrocarbon receptor (AHR): “pioneer member” of the basic-helix/loop/helix per - Arnt - sim (bHLH/PAS) family of “sensors” of foreign and endogenous signals
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- The New Aryl Hydrocarbon Receptor Antagonist E/Z-2-Benzylindene-5,6-Dimethoxy-3,3-Dimethylindan-1-One Protects against UVB-Induced Signal Transduction
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- Exactly the Same but Different: Promiscuity and Diversity in the Molecular Mechanisms of Action of the Aryl Hydrocarbon (Dioxin) Receptor
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- Nucleotide Excision Repair Driven by the Dissociation of CAK from TFIIH
- (2008) Frédéric Coin et al. MOLECULAR CELL
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