A novel form of necrosis, TRIAD, occurs in human Huntington’s disease

Targeting TEAD/YAP-transcription-dependent necrosis, TRIAD, ameliorates Huntington’s disease pathology

(2016) Ying Mao et al.   HUMAN MOLECULAR GENETICS

The hnRNP-Htt axis regulates necrotic cell death induced by transcriptional repression through impaired RNA splicing

(2016) Y Mao et al.   Cell Death & Disease

c-Abl antagonizes the YAP oncogenic function

(2014) R Keshet et al.   CELL DEATH AND DIFFERENTIATION

Rapamycin increases survival in ALS mice lacking mature lymphocytes

(2013) Kim A Staats et al.   Molecular Neurodegeneration

Rapamycin treatment augments motor neuron degeneration in SOD1G93Amouse model of amyotrophic lateral sclerosis

(2011) Xiaojie Zhang et al.   Autophagy

Polo-like kinases mediate cell survival in mitochondrial dysfunction

(2009) T. Matsumoto et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Progressive decrease in the level of YAPdeltaCs, prosurvival isoforms of YAP, in the spinal cord of transgenic mouse carrying a mutantSOD1gene

(2008) Nobutoshi Morimoto et al.   JOURNAL OF NEUROSCIENCE RESEARCH