Tumor-derived exosomes educate dendritic cells to promote tumor metastasis via HSP72/HSP105-TLR2/TLR4 pathway
出版年份 2017 全文链接
标题
Tumor-derived exosomes educate dendritic cells to promote tumor metastasis via HSP72/HSP105-TLR2/TLR4 pathway
作者
关键词
-
出版物
OncoImmunology
Volume 6, Issue 12, Pages e1362527
出版商
Informa UK Limited
发表日期
2017-08-18
DOI
10.1080/2162402x.2017.1362527
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Communication by Extracellular Vesicles: Where We Are and Where We Need to Go
- (2016) Mercedes Tkach et al. CELL
- Tumor-derived exosomes elicit tumor suppression in murine hepatocellular carcinoma models and humansin vitro
- (2016) Quan Rao et al. HEPATOLOGY
- Exosomes and tumor-mediated immune suppression
- (2016) Theresa L. Whiteside JOURNAL OF CLINICAL INVESTIGATION
- Satb1 Overexpression Drives Tumor-Promoting Activities in Cancer-Associated Dendritic Cells
- (2016) Amelia J. Tesone et al. Cell Reports
- Glypican-1 identifies cancer exosomes and detects early pancreatic cancer
- (2015) Sonia A. Melo et al. NATURE
- Pancreatic cancer exosomes initiate pre-metastatic niche formation in the liver
- (2015) Bruno Costa-Silva et al. NATURE CELL BIOLOGY
- The regulation of cancer cell migration by lung cancer cell-derived exosomes through TGF-β and IL-10
- (2015) YUZHOU WANG et al. Oncology Letters
- The exosomes in tumor immunity
- (2015) Yanfang Liu et al. OncoImmunology
- The extent to which melanoma alters tissue-resident dendritic cell function correlates with tumorigenicity
- (2015) Kristian Michael Hargadon OncoImmunology
- Human Tumor-Derived Exosomes Down-Modulate NKG2D Expression
- (2014) A. Clayton et al. JOURNAL OF IMMUNOLOGY
- LPLUNC1 suppresses IL-6-induced nasopharyngeal carcinoma cell proliferation via inhibiting the Stat3 activation
- (2013) Q Liao et al. ONCOGENE
- Immunosuppressive exosomes from TGF-β1 gene-modified dendritic cells attenuate Th17-mediated inflammatory autoimmune disease by inducing regulatory T cells
- (2011) Zhijian Cai et al. CELL RESEARCH
- IL-6 Increases MMP-13 Expression and Motility in Human Chondrosarcoma Cells
- (2011) Chih-Hsin Tang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Chemokine-Containing Exosomes Are Released from Heat-Stressed Tumor Cells via Lipid Raft-Dependent Pathway and Act as Efficient Tumor Vaccine
- (2011) T. Chen et al. JOURNAL OF IMMUNOLOGY
- IL-17 induces AKT-dependent IL-6/JAK2/STAT3 activation and tumor progression in hepatocellular carcinoma
- (2011) Fang-Ming Gu et al. Molecular Cancer
- Membrane-associated Hsp72 from tumor-derived exosomes mediates STAT3-dependent immunosuppressive function of mouse and human myeloid-derived suppressor cells
- (2010) Fanny Chalmin et al. JOURNAL OF CLINICAL INVESTIGATION
- IL-6 promotion of glioblastoma cell invasion and angiogenesis in U251 and T98G cell lines
- (2010) Qinglin Liu et al. JOURNAL OF NEURO-ONCOLOGY
- Genetic deletion of mPGES-1 abolishes PGE2 production in murine dendritic cells and alters the cytokine profile, but does not affect maturation or migration
- (2010) S.U. Monrad et al. PROSTAGLANDINS LEUKOTRIENES AND ESSENTIAL FATTY ACIDS
- Hsp105β upregulates hsp70 gene expression through signal transducer and activator of transcription-3
- (2009) Nobuyuki Yamagishi et al. FEBS Journal
- Activation of hepatocytes by extracellular heat shock protein 72
- (2008) Elizabeth Galloway et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Bone morphogenetic protein-2 enhances the motility of chondrosarcoma cells via activation of matrix metalloproteinase-13
- (2008) Chun-Han Hou et al. BONE
- Induction of myeloid-derived suppressor cells by tumor exosomes
- (2008) Xiaoyu Xiang et al. INTERNATIONAL JOURNAL OF CANCER
- Rapamycin suppresses TLR4-triggered IL-6 and PGE2 production of colon cancer cells by inhibiting TLR4 expression and NF-κB activation
- (2008) Qiaoling Sun et al. MOLECULAR IMMUNOLOGY
Become a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get StartedAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started