Chloroquine triggers Epstein-Barr virus replication through phosphorylation of KAP1/TRIM28 in Burkitt lymphoma cells

Chloroquine inhibits lytic replication of Kaposi's sarcoma-associated herpesvirus by disrupting mTOR and p38-MAPK activation

(2016) Mengtian Yang et al.   ANTIVIRAL RESEARCH

Activated Nrf2 Interacts with Kaposi's Sarcoma-Associated Herpesvirus Latency Protein LANA-1 and Host Protein KAP1 To Mediate Global Lytic Gene Repression

(2015) Olsi Gjyshi et al.   JOURNAL OF VIROLOGY

STAT3 Regulates Lytic Activation of Kaposi's Sarcoma-Associated Herpesvirus

(2015) Christine A. King et al.   JOURNAL OF VIROLOGY

LMP1-Induced Sumoylation Influences the Maintenance of Epstein-Barr Virus Latency through KAP1

(2015) Gretchen L. Bentz et al.   JOURNAL OF VIROLOGY

Release of human cytomegalovirus from latency by a KAP1/TRIM28 phosphorylation switch

(2015) Benjamin Rauwel et al.   eLife

DNA Damage Signaling Is Induced in the Absence of Epstein—Barr Virus (EBV) Lytic DNA Replication and in Response to Expression of ZEBRA

(2015) Ruth Wang'ondu et al.   PLoS One

Kaposi's Sarcoma-Associated Herpesvirus-Encoded LANA Interacts with Host KAP1 To Facilitate Establishment of Viral Latency

(2014) R. Sun et al.   JOURNAL OF VIROLOGY

DNA double-strand breaks promote methylation of histone H3 on lysine 9 and transient formation of repressive chromatin

(2014) M. K. Ayrapetov et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Signal Transducer and Activator of Transcription 3 Limits Epstein-Barr Virus Lytic Activation in B Lymphocytes

(2013) E. R. Hill et al.   JOURNAL OF VIROLOGY

B Lymphocytes from Patients with a Hypomorphic Mutation in STAT3 Resist Epstein-Barr Virus-Driven Cell Proliferation

(2013) S. Koganti et al.   JOURNAL OF VIROLOGY

Kaposi's Sarcoma-Associated Herpesvirus Kaposin B Induces Unique Monophosphorylation of STAT3 at Serine 727 and MK2-Mediated Inactivation of the STAT3 Transcriptional Repressor TRIM28

(2013) C. A. King   JOURNAL OF VIROLOGY

Krüppel-associated Box (KRAB)-associated Co-repressor (KAP-1) Ser-473 Phosphorylation Regulates Heterochromatin Protein 1β (HP1-β) Mobilization and DNA Repair in Heterochromatin

(2012) Emma Bolderson et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

The Cellular Ataxia Telangiectasia-Mutated Kinase Promotes Epstein-Barr Virus Lytic Reactivation in Response to Multiple Different Types of Lytic Reactivation-Inducing Stimuli

(2012) S. R. Hagemeier et al.   JOURNAL OF VIROLOGY

KAP1 Protein: An Enigmatic Master Regulator of the Genome

(2011) Sushma Iyengar et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Patterns of chloroquine use and resistance in sub-Saharan Africa: a systematic review of household survey and molecular data

(2011) Anne EP Frosch et al.   MALARIA JOURNAL

The ATM Substrate KAP1 Controls DNA Repair in Heterochromatin: Regulation by HP1 Proteins and Serine 473/824 Phosphorylation

(2011) D. White et al.   MOLECULAR CANCER RESEARCH

The effects of Plasmodium falciparum malaria on immune control of B lymphocytes in Gambian children

(2010) H. C. WHITTLE et al.   CLINICAL AND EXPERIMENTAL IMMUNOLOGY

Kruppel-Associated Box Domain-Associated Protein-1 as a Latency Regulator for Kaposi's Sarcoma-Associated Herpesvirus and Its Modulation by the Viral Protein Kinase

(2009) P.-C. Chang et al.   CANCER RESEARCH

Upregulation of STAT3 Marks Burkitt Lymphoma Cells Refractory to Epstein-Barr Virus Lytic Cycle Induction by HDAC Inhibitors

(2009) D. Daigle et al.   JOURNAL OF VIROLOGY

DNA Damage Responses: Mechanisms and Roles in Human Disease: 2007 G.H.A. Clowes Memorial Award Lecture

(2008) M. B. Kastan   MOLECULAR CANCER RESEARCH

ATM Signaling Facilitates Repair of DNA Double-Strand Breaks Associated with Heterochromatin

(2008) Aaron A. Goodarzi et al.   MOLECULAR CELL

Analyzing real-time PCR data by the comparative CT method

(2008) Thomas D Schmittgen et al.   Nature Protocols

Targeting lysosomal degradation induces p53-dependent cell death and prevents cancer in mouse models of lymphomagenesis

(2007) Kirsteen H. Maclean et al.   JOURNAL OF CLINICAL INVESTIGATION