Hippocampal calpain is required for the consolidation and reconsolidation but not extinction of contextual fear memory
出版年份 2017 全文链接
标题
Hippocampal calpain is required for the consolidation and reconsolidation but not extinction of contextual fear memory
作者
关键词
Calpain, Hippocampus, Fear conditioning, ALLN, c-fos
出版物
Molecular Brain
Volume 10, Issue 1, Pages -
出版商
Springer Nature
发表日期
2017-12-19
DOI
10.1186/s13041-017-0341-8
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Functional Connectivity of Multiple Brain Regions Required for the Consolidation of Social Recognition Memory
- (2017) Toshiyuki Tanimizu et al. JOURNAL OF NEUROSCIENCE
- Brain networks activated to form object recognition memory
- (2017) Toshiyuki Tanimizu et al. BRAIN RESEARCH BULLETIN
- Sex differences in hippocampal function
- (2016) Wendy A. Koss et al. JOURNAL OF NEUROSCIENCE RESEARCH
- N-glycosylation in the hippocampus is required for the consolidation and reconsolidation of contextual fear memory
- (2016) Hiroyoshi Inaba et al. NEUROBIOLOGY OF LEARNING AND MEMORY
- Calpain-1 and Calpain-2: The Yin and Yang of Synaptic Plasticity and Neurodegeneration
- (2016) Michel Baudry et al. TRENDS IN NEUROSCIENCES
- Effect of context exposure after fear learning on memory generalization in mice
- (2016) Ayano Fujinaka et al. Molecular Brain
- PARP-1 activity is required for the reconsolidation and extinction of contextual fear memory
- (2015) Hiroyoshi Inaba et al. Molecular Brain
- Memory Enhancement by Targeting Cdk5 Regulation of NR2B
- (2014) Florian Plattner et al. NEURON
- Cdk5/p35 functions as a crucial regulator of spatial learning and memory
- (2014) Tomohide Mishiba et al. Molecular Brain
- Enhancement of fear memory by retrieval through reconsolidation
- (2014) Hotaka Fukushima et al. eLife
- Targeting calpain in synaptic plasticity
- (2013) Michel Baudry et al. EXPERT OPINION ON THERAPEUTIC TARGETS
- Conditional Disruption of Calpain in the CNS Alters Dendrite Morphology, Impairs LTP, and Promotes Neuronal Survival following Injury
- (2013) M. Amini et al. JOURNAL OF NEUROSCIENCE
- A cellular model of memory reconsolidation involves reactivation-induced destabilization and restabilization at the sensorimotor synapse in Aplysia
- (2012) S.-H. Lee et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Cyclin-Dependent Kinase 5 Activator p25 Is Generated During Memory Formation and Is Reduced at an Early Stage in Alzheimer's Disease
- (2011) Olivia Engmann et al. BIOLOGICAL PSYCHIATRY
- Protein Degradation during Reconsolidation as a Mechanism for Memory Reorganization
- (2011) Bong-Kiun Kaang et al. Frontiers in Behavioral Neuroscience
- Induction and requirement of gene expression in the anterior cingulate cortex and medial prefrontal cortex for the consolidation of inhibitory avoidance memory
- (2011) Yue Zhang et al. Molecular Brain
- Protein degradation and memory formation
- (2010) Diasynou Fioravante et al. BRAIN RESEARCH BULLETIN
- Brain-Derived Neurotrophic Factor and Epidermal Growth Factor Activate Neuronal m-Calpain via Mitogen-Activated Protein Kinase-Dependent Phosphorylation
- (2010) S. Zadran et al. JOURNAL OF NEUROSCIENCE
- Regulation of Calpain-2 in Neurons: Implications for Synaptic Plasticity
- (2010) Sohila Zadran et al. MOLECULAR NEUROBIOLOGY
- Towards a molecular understanding of sex differences in memory formation
- (2010) Keiko Mizuno et al. TRENDS IN NEUROSCIENCES
- Brain Region-Specific Gene Expression Activation Required for Reconsolidation and Extinction of Contextual Fear Memory
- (2009) N. Mamiya et al. JOURNAL OF NEUROSCIENCE
- Activation of LVGCCs and CB1 receptors required for destabilization of reactivated contextual fear memories
- (2008) A. Suzuki et al. LEARNING & MEMORY
- Synaptic Protein Degradation Underlies Destabilization of Retrieved Fear Memory
- (2008) S.-H. Lee et al. SCIENCE
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