Allergic sensitization is associated with inadequate antioxidant responses in mice and men

Disruption of the Transcription Factor Nrf2 Promotes Pro-Oxidative Dendritic Cells That Stimulate Th2-Like Immunoresponsiveness upon Activation by Ambient Particulate Matter

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Nuclear Factor-erythroid 2 (NF-E2) p45-related Factor-2 (Nrf2) Modulates Dendritic Cell Immune Function through Regulation of p38 MAPK-cAMP-responsive Element Binding Protein/Activating Transcription Factor 1 Signaling

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Inhaled Birch Pollen Extract Induces Airway Hyperresponsiveness via Oxidative Stress but Independently of Pollen-Intrinsic NADPH Oxidase Activity, or the TLR4-TRIF Pathway

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Distinct TLR-mediated pathways regulate house dust mite–induced allergic disease in the upper and lower airways

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Low CXCL13 Expression, Splenic Lymphoid Tissue Atrophy and Germinal Center Disruption in Severe Canine Visceral Leishmaniasis

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Persistent Activation of Dendritic Cells after Resolution of Allergic Airway Inflammation Breaks Tolerance to Inhaled Allergens in Mice

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DAMPs and PDT-mediated photo-oxidative stress: exploring the unknown

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The composition of house dust mite is critical for mucosal barrier dysfunction and allergic sensitisation

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Dynamics in cytokine responses during the development of occupational sensitization to rats

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Pattern Recognition Receptors and Inflammation

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Pollen-Induced Oxidative Stress Influences Both Innate and Adaptive Immune Responses via Altering Dendritic Cell Functions

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Complement-mediated regulation of the IL-17A axis is a central genetic determinant of the severity of experimental allergic asthma

(2010) Stephane Lajoie et al.   NATURE IMMUNOLOGY

The T helper type 2 response to cysteine proteases requires dendritic cell–basophil cooperation via ROS-mediated signaling

(2010) Hua Tang et al.   NATURE IMMUNOLOGY

Toll/IL-1 Signaling Is Critical for House Dust Mite–specific Th1 and Th2 Responses

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A protective role of nuclear factor-erythroid 2-related factor-2 (Nrf2) in inflammatory disorders

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House dust mite allergen induces asthma via Toll-like receptor 4 triggering of airway structural cells

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Ozone inhalation induces exacerbation of eosinophilic airway inflammation and hyperresponsiveness in allergen-sensitized mice

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Suppression by CD4+CD25+ Regulatory T Cells Is Dependent on Expression of Heme Oxygenase-1 in Antigen-Presenting Cells

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