Glycyrrhizic acid exerts anti-inflammatory effect to improve cerebral vasospasm secondary to subarachnoid hemorrhage in a rat model
出版年份 2017 全文链接
标题
Glycyrrhizic acid exerts anti-inflammatory effect to improve cerebral vasospasm secondary to subarachnoid hemorrhage in a rat model
作者
关键词
-
出版物
NEUROLOGICAL RESEARCH
Volume 39, Issue 8, Pages 727-732
出版商
Informa UK Limited
发表日期
2017-04-18
DOI
10.1080/01616412.2017.1316903
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Expression of high-mobility group box-1 (HMGB1) in the basilar artery after experimental subarachnoid hemorrhage
- (2016) Xu Dong Zhao et al. JOURNAL OF CLINICAL NEUROSCIENCE
- Unknown
- (2016) JOURNAL OF INTERNAL MEDICINE
- Unknown
- (2016) JOURNAL OF NEUROSCIENCE RESEARCH
- 4′-O-β-d-glucosyl-5-O-methylvisamminol, an active ingredient of Saposhnikovia divaricata, attenuates high-mobility group box 1 and subarachnoid hemorrhage-induced vasospasm in a rat model
- (2015) Chih-Zen Chang et al. Behavioral and Brain Functions
- Fisetin alleviates early brain injury following experimental subarachnoid hemorrhage in rats possibly by suppressing TLR 4/NF-κB signaling pathway
- (2015) Chen-hui Zhou et al. BRAIN RESEARCH
- Biomarker Discovery in Cerebral Vasospasm after Aneurysmal Subarachnoid Hemorrhage
- (2015) Magdalena M. Przybycien-Szymanska et al. Journal of Stroke & Cerebrovascular Diseases
- Glycyrrhizin Attenuates Proinflammatory Cytokines through a Peroxisome Proliferator-Activated Receptor-γ-Dependent Mechanism and Experimental Vasospasm in a Rat Model
- (2015) Chih-Zen Chang et al. JOURNAL OF VASCULAR RESEARCH
- Biomarker Discovery in Cerebral Vasospasm after Aneurysmal Subarachnoid Hemorrhage
- (2015) Magdalena M. Przybycien-Szymanska et al. Journal of Stroke & Cerebrovascular Diseases
- Dynamic expression of the suppressor of cytokine signaling-3 and cytokines in the cerebral basilar artery of rats with subarachnoid hemorrhage, and the effect of acetylcholine
- (2014) Jin-Ning Song et al. ACTA NEUROCHIRURGICA
- Early release of high-mobility group box 1 (HMGB1) from neurons in experimental subarachnoid hemorrhage in vivo and in vitro
- (2014) Qing Sun et al. Journal of Neuroinflammation
- Unknown
- (2014) Melinda Magna et al. MOLECULAR MEDICINE
- Glycyrrhizin Attenuates Toll Like Receptor-2, -4 and Experimental Vasospasm in a Rat Model
- (2014) Chih-Zen Chang et al. Journal of Immunology Research
- Glycyrrhizic acid confers neuroprotection after subarachnoid hemorrhage via inhibition of high mobility group box-1 protein: A hypothesis for novel therapy of subarachnoid hemorrhage
- (2013) Qing Sun et al. MEDICAL HYPOTHESES
- HMGB1 Is a Therapeutic Target for Sterile Inflammation and Infection
- (2011) Ulf Andersson et al. Annual Review of Immunology
- Relationship between brain interstitial fluid tumor necrosis factor-α and cerebral vasospasm after aneurysmal subarachnoid hemorrhage
- (2010) Khalid A. Hanafy et al. JOURNAL OF CLINICAL NEUROSCIENCE
- Do Current Animal Models of Intracerebral Hemorrhage Mirror the Human Pathology?
- (2010) Opeolu Adeoye et al. Translational Stroke Research
- Subarachnoid Hemorrhage Induces Gliosis and Increased Expression of the Pro-inflammatory Cytokine High Mobility Group Box 1 Protein
- (2010) Kentaro Murakami et al. Translational Stroke Research
- Neutralization of Interleukin-1β Reduces Vasospasm and Alters Cerebral Blood Vessel Density Following Experimental Subarachnoid Hemorrhage in Rats
- (2009) Halina Jedrzejowska-Szypulka et al. CURRENT NEUROVASCULAR RESEARCH
- Tumor Necrosis Factor-α Mediates Hemolysis-Induced Vasoconstriction and the Cerebral Vasospasm Evoked by Subarachnoid Hemorrhage
- (2009) Carmine Vecchione et al. HYPERTENSION
- Curcumin Attenuates Vascular Inflammation and Cerebral Vasospasm After Subarachnoid Hemorrhage in Mice
- (2008) Chandramohan Wakade et al. ANTIOXIDANTS & REDOX SIGNALING
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More