Promoter-bound METTL3 maintains myeloid leukaemia by m6A-dependent translation control

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RNA fate determination through cotranscriptional adenosine methylation and microprocessor binding

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A CRISPR Dropout Screen Identifies Genetic Vulnerabilities and Therapeutic Targets in Acute Myeloid Leukemia

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Highly Efficient Genome Editing of Murine and Human Hematopoietic Progenitor Cells by CRISPR/Cas9

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The heteromeric transcription factor GABP activates the ITGAM/CD11b promoter and induces myeloid differentiation

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5′ UTR m6A Promotes Cap-Independent Translation

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N6-methyladenosine Modulates Messenger RNA Translation Efficiency

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Discovery of cancer drug targets by CRISPR-Cas9 screening of protein domains

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m6A RNA Modification Controls Cell Fate Transition in Mammalian Embryonic Stem Cells

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BET protein inhibition shows efficacy against JAK2V617F-driven neoplasms

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A METTL3–METTL14 complex mediates mammalian nuclear RNA N6-adenosine methylation

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Transcriptome-wide mapping of N6-methyladenosine by m6A-seq based on immunocapturing and massively parallel sequencing

(2013) Dan Dominissini et al.   Nature Protocols

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Insights into RNA Biology from an Atlas of Mammalian mRNA-Binding Proteins

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(2012) Dan Dominissini et al.   NATURE

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Symmetric dimethylation of H3R2 is a newly identified histone mark that supports euchromatin maintenance

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Inhibition of BET recruitment to chromatin as an effective treatment for MLL-fusion leukaemia

(2011) Mark A. Dawson et al.   NATURE

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Simple Combinations of Lineage-Determining Transcription Factors Prime cis-Regulatory Elements Required for Macrophage and B Cell Identities

(2010) Sven Heinz et al.   MOLECULAR CELL

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(2010) Cole Trapnell et al.   NATURE BIOTECHNOLOGY

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JAK2 phosphorylates histone H3Y41 and excludes HP1α from chromatin

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