Role of nerve-muscle interactions and reactive oxygen species in regulation of muscle proteostasis with ageing
出版年份 2017 全文链接
标题
Role of nerve-muscle interactions and reactive oxygen species in regulation of muscle proteostasis with ageing
作者
关键词
-
出版物
JOURNAL OF PHYSIOLOGY-LONDON
Volume 595, Issue 20, Pages 6409-6415
出版商
Wiley
发表日期
2017-08-09
DOI
10.1113/jp274336
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Happily (n)ever after: Aging in the context of oxidative stress, proteostasis loss and cellular senescence
- (2017) Annika Höhn et al. Redox Biology
- A new mouse model of frailty: the Cu/Zn superoxide dismutase knockout mouse
- (2017) Sathyaseelan S. Deepa et al. GeroScience
- The effect of lengthening contractions on neuromuscular junction structure in adult and old mice
- (2016) Aphrodite Vasilaki et al. AGE
- Chaperone mediated autophagy in aging: Starve to prosper
- (2016) Maria Xilouri et al. AGEING RESEARCH REVIEWS
- Mechanisms of protein balance in skeletal muscle
- (2016) T.G. Anthony DOMESTIC ANIMAL ENDOCRINOLOGY
- Age-dependent motor unit remodelling in human limb muscles
- (2015) Mathew Piasecki et al. BIOGERONTOLOGY
- Age-related neuromuscular changes affecting human vastus lateralis
- (2015) M. Piasecki et al. JOURNAL OF PHYSIOLOGY-LONDON
- Proteostasis and aging
- (2015) Susmita Kaushik et al. NATURE MEDICINE
- Neuron specific reduction in CuZnSOD is not sufficient to initiate a full sarcopenia phenotype
- (2015) Kavithalakshmi Sataranatarajan et al. Redox Biology
- Aging increases the oxidation of dichlorohydrofluorescein in single isolated skeletal muscle fibers at rest, but not during contractions
- (2013) Jesus Palomero et al. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY
- Chaperone-mediated autophagy: roles in disease and aging
- (2013) Ana Maria Cuervo et al. CELL RESEARCH
- CuZnSOD gene deletion targeted to skeletal muscle leads to loss of contractile force but does not cause muscle atrophy in adult mice
- (2013) Yiqiang Zhang et al. FASEB JOURNAL
- Neuron-specific expression of CuZnSOD prevents the loss of muscle mass and function that occurs in homozygous CuZnSOD-knockout mice
- (2013) Giorgos K. Sakellariou et al. FASEB JOURNAL
- The Role of Oxidative Stress in Nervous System Aging
- (2013) Catrina Sims-Robinson et al. PLoS One
- Skeletal muscle weakness due to deficiency of CuZn-superoxide dismutase is associated with loss of functional innervation
- (2011) Lisa M. Larkin et al. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY
- Striking Denervation of Neuromuscular Junctions without Lumbar Motoneuron Loss in Geriatric Mouse Muscle
- (2011) Ruth Jinfen Chai et al. PLoS One
- The age-related failure of adaptive responses to contractile activity in skeletal muscle is mimicked in young mice by deletion of Cu,Zn superoxide dismutase
- (2010) Aphrodite Vasilaki et al. AGING CELL
- Age-associated alterations of the neuromuscular junction
- (2010) Youngmok C. Jang et al. EXPERIMENTAL GERONTOLOGY
- Attenuation of age-related changes in mouse neuromuscular synapses by caloric restriction and exercise
- (2010) G. Valdez et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Sarcopenia of aging: Underlying cellular mechanisms and protection by calorie restriction
- (2009) Emanuele Marzetti et al. BIOFACTORS
- Increased superoxide in vivo accelerates age-associated muscle atrophy through mitochondrial dysfunction and neuromuscular junction degeneration
- (2009) Youngmok C. Jang et al. FASEB JOURNAL
- Exercise-Induced Oxidative Stress: Cellular Mechanisms and Impact on Muscle Force Production
- (2008) Scott K. Powers et al. PHYSIOLOGICAL REVIEWS
- Moderate exercise is an antioxidant: Upregulation of antioxidant genes by training
- (2007) Mari-Carmen Gomez-Cabrera et al. FREE RADICAL BIOLOGY AND MEDICINE
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started