miR-30d-5p Plays an Important Role in Autophagy and Apoptosis in Developing Rat Brains After Hypoxic–Ischemic Injury
出版年份 2017 全文链接
标题
miR-30d-5p Plays an Important Role in Autophagy and Apoptosis in Developing Rat Brains After Hypoxic–Ischemic Injury
作者
关键词
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出版物
JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY
Volume 76, Issue 8, Pages 709-719
出版商
Oxford University Press (OUP)
发表日期
2017-07-22
DOI
10.1093/jnen/nlx052
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- MiR-193b promotes autophagy and non-apoptotic cell death in oesophageal cancer cells
- (2016) Michelle J. Nyhan et al. BMC CANCER
- MicroRNA-155 suppresses autophagy in chondrocytes by modulating expression of autophagy proteins
- (2016) S. D'Adamo et al. OSTEOARTHRITIS AND CARTILAGE
- Toward the promise of microRNAs – Enhancing reproducibility and rigor in microRNA research
- (2016) Kenneth W. Witwer et al. RNA Biology
- Ischemia-induced autophagy contributes to neurodegeneration in cerebellar Purkinje cells in the developing rat brain and in primary cortical neurons in vitro
- (2015) Alicia K. Au et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE
- Re-thinking miRNA-mRNA interactions: Intertwining issues confound target discovery
- (2015) Nicole Cloonan BIOESSAYS
- Genome–wide microRNA expression profiling in placentas from pregnant women exposed to BPA
- (2015) Bruna De Felice et al. BMC Medical Genomics
- Neuronal KATP channels mediate hypoxic preconditioning and reduce subsequent neonatal hypoxic–ischemic brain injury
- (2015) Hong-Shuo Sun et al. EXPERIMENTAL NEUROLOGY
- Integrated and comparative miRNA analysis of starvation-induced autophagy in mouse embryonic fibroblasts
- (2015) Jianzhou Cui et al. GENE
- Sparse Modeling Reveals miRNA Signatures for Diagnostics of Inflammatory Bowel Disease
- (2015) Matthias Hübenthal et al. PLoS One
- Ischemic Preconditioning Mediates Neuroprotection against Ischemia in Mouse Hippocampal CA1 Neurons by Inducing Autophagy
- (2015) Chunlin Gao et al. PLoS One
- Altered microRNA profiles in cerebrospinal fluid exosome in Parkinson disease and Alzheimer disease
- (2015) YaXing Gui et al. Oncotarget
- Microarray Profiling and Co-Expression Network Analysis of LncRNAs and mRNAs in Neonatal Rats Following Hypoxic-ischemic Brain Damage
- (2015) Fengyan Zhao et al. Scientific Reports
- Neuronal injury in rat model of permanent focal cerebral ischemia is associated with activation of autophagic and lysosomal pathways
- (2014) Ya-Dan Wen et al. Autophagy
- Autophagic neuron death in neonatal brain ischemia/hypoxia
- (2014) Yasuo Uchiyama et al. Autophagy
- w007B protects brain against ischemia–reperfusion injury in rats through inhibiting inflammation, apoptosis and autophagy
- (2014) Qixin Bu et al. BRAIN RESEARCH
- Increased autophagy reduces endoplasmic reticulum stress after neonatal hypoxia–ischemia: Role of protein synthesis and autophagic pathways
- (2014) Silvia Carloni et al. EXPERIMENTAL NEUROLOGY
- Pinocembrin Protects the Brain against Ischemia-Reperfusion Injury and Reverses the Autophagy Dysfunction in the Penumbra Area
- (2014) Gang Zhao et al. MOLECULES
- Down-Regulation of miRNA-30a Alleviates Cerebral Ischemic Injury Through Enhancing Beclin 1-Mediated Autophagy
- (2014) Peng Wang et al. NEUROCHEMICAL RESEARCH
- Recombinant Human Erythropoietin Improves the Neurofunctional Recovery of Rats Following Traumatic Brain Injury via an Increase in Circulating Endothelial Progenitor Cells
- (2014) Liang Wang et al. Translational Stroke Research
- mir-30d Regulates multiple genes in the autophagy pathway and impairs autophagy process in human cancer cells
- (2013) Xiaojun Yang et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- MiR-30d induces apoptosis and is regulated by the Akt/FOXO pathway in renal cell carcinoma
- (2013) Chenglin Wu et al. CELLULAR SIGNALLING
- Nelfinavir Inhibits Intra-Mitochondrial Calcium Influx and Protects Brain against Hypoxic-Ischemic Injury in Neonatal Mice
- (2013) Irina V. Utkina-Sosunova et al. PLoS One
- Autophagosome formation—The role of ULK1 and Beclin1–PI3KC3 complexes in setting the stage
- (2013) Martina Wirth et al. SEMINARS IN CANCER BIOLOGY
- miRNA-130a Targets ATG2B and DICER1 to Inhibit Autophagy and Trigger Killing of Chronic Lymphocytic Leukemia Cells
- (2012) V. Kovaleva et al. CANCER RESEARCH
- Enhanced NMDA receptor tyrosine phosphorylation and increased brain injury following neonatal hypoxia–ischemia in mice with neuronal Fyn overexpression
- (2012) Renatta Knox et al. NEUROBIOLOGY OF DISEASE
- The neuroprotective role of TERT via an antiapoptotic mechanism in neonatal rats after hypoxia–ischemia brain injury
- (2012) Fengyan Zhao et al. NEUROSCIENCE LETTERS
- miRNA-Mediated Gene Silencing by Translational Repression Followed by mRNA Deadenylation and Decay
- (2012) S. Djuranovic et al. SCIENCE
- The role of oxidative stress in perinatal hypoxic-ischemic brain injury
- (2012) Brankica Vasiljevic et al. Srpski Arhiv za Celokupno Lekarstvo
- Activation of autophagy and Akt/CREB signaling play an equivalent role in the neuroprotective effect of rapamycin in neonatal hypoxia-ischemia
- (2010) Silvia Carloni et al. Autophagy
- Autophagy in the Pathogenesis of Disease
- (2008) Beth Levine et al. CELL
- Regulation of the Mammalian Nervous System by MicroRNAs
- (2008) Y. Zeng MOLECULAR PHARMACOLOGY
- Protective role of autophagy in neonatal hypoxia–ischemia induced brain injury
- (2008) Silvia Carloni et al. NEUROBIOLOGY OF DISEASE
- Impact of indolent inflammation on neonatal hypoxic-ischemic brain injury in mice
- (2007) John D.E. Barks et al. INTERNATIONAL JOURNAL OF DEVELOPMENTAL NEUROSCIENCE
- Focal cerebral ischemia induces upregulation of Beclin 1 and autophagy-like cell death
- (2007) A. Rami et al. NEUROBIOLOGY OF DISEASE
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