Par-aPKC-dependent and -independent mechanisms cooperatively control cell polarity, Hippo signaling, and cell positioning in 16-cell stage mouse embryos
出版年份 2015 全文链接
标题
Par-aPKC-dependent and -independent mechanisms cooperatively control cell polarity, Hippo signaling, and cell positioning in 16-cell stage mouse embryos
作者
关键词
-
出版物
DEVELOPMENT GROWTH & DIFFERENTIATION
Volume 57, Issue 8, Pages 544-556
出版商
Wiley
发表日期
2015-10-09
DOI
10.1111/dgd.12235
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Position- and polarity-dependent Hippo signaling regulates cell fates in preimplantation mouse embryos
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- Notch and Hippo Converge on Cdx2 to Specify the Trophectoderm Lineage in the Mouse Blastocyst
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- Angiomotins link F-actin architecture to Hippo pathway signaling
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- HIPPO Pathway Members Restrict SOX2 to the Inner Cell Mass Where It Promotes ICM Fates in the Mouse Blastocyst
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- Modulating F-actin organization induces organ growth by affecting the Hippo pathway
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- Cell Polarity Regulator PARD6B Is Essential for Trophectoderm Formation in the Preimplantation Mouse Embryo1
- (2010) Vernadeth B. Alarcon BIOLOGY OF REPRODUCTION
- Mechanisms of trophectoderm fate specification in preimplantation mouse development
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- The Hippo Signaling Pathway Components Lats and Yap Pattern Tead4 Activity to Distinguish Mouse Trophectoderm from Inner Cell Mass
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- Inactivation of aPKCλ Reveals a Context Dependent Allocation of Cell Lineages in Preimplantation Mouse Embryos
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- Blastomeres of the mouse embryo lose totipotency after the fifth cleavage division: Expression of Cdx2 and Oct4 and developmental potential of inner and outer blastomeres of 16- and 32-cell embryos
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- Cdx2 acts downstream of cell polarization to cell-autonomously promote trophectoderm fate in the early mouse embryo
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