期刊
JOURNAL OF EXPERIMENTAL BOTANY
卷 69, 期 6, 页码 1325-1333出版社
OXFORD UNIV PRESS
DOI: 10.1093/jxb/erx425
关键词
Autophagy; biotroph; hypersensitive response; innate immunity; Joka2; necrotroph; NLR; PCD; TOR; virus
资金
- Biotechnology and Biological Sciences Research Council [BB/M002462/1, BB/M011224/1]
- BBSRC doctoral training programme
- Royal Thai Government scholarship
- Imperial College Schrodinger Scholarship Scheme
- TUBITAK
- British Council Newton-Katip Celebi Fund
- Consejo Nacional de Investigaciones Cientificas y Tecnicas (CONICET)
- BBSRC [BB/M002462/1, 1655056] Funding Source: UKRI
- Biotechnology and Biological Sciences Research Council [1655056, BB/M002462/1] Funding Source: researchfish
In plants, the highly conserved catabolic process of autophagy has long been known as a means of maintaining cellular homeostasis and coping with abiotic stress conditions. Accumulating evidence has linked autophagy to immunity against invading pathogens, regulating plant cell death, and antimicrobial defences. In turn, it appears that phytopathogens have evolved ways not only to evade autophagic clearance but also to modulate and co-opt autophagy for their own benefit. In this review, we summarize and discuss the emerging discoveries concerning how pathogens modulate both host and self-autophagy machineries to colonize their host plants, delving into the arms race that determines the fate of interorganismal interaction.
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