Inactivation of ferric uptake regulator (Fur) attenuates Helicobacter pylori J99 motility by disturbing the flagellar motor switch and autoinducer-2 production

The Helicobacter pylori CZB Cytoplasmic Chemoreceptor TlpD Forms an Autonomous Polar Chemotaxis Signaling Complex That Mediates a Tactic Response to Oxidative Stress

(2016) Kieran D. Collins et al.   JOURNAL OF BACTERIOLOGY

Helicobacter pylori infection: An overview of bacterial virulence factors and pathogenesis

(2016) Cheng-Yen Kao et al.   Biomedical Journal

CsrA RegulatesHelicobacter pyloriJ99 Motility and Adhesion by Controlling Flagella Formation

(2014) Cheng-Yen Kao et al.   HELICOBACTER

Higher Motility Enhances Bacterial Density and Inflammatory Response in Dyspeptic Patients Infected withHelicobacter pylori

(2012) Cheng-Yen Kao et al.   HELICOBACTER

Length of Thymidine Homopolymeric Repeats Modulates Promoter Activity of sabA in Helicobacter pylori

(2012) Cheng-Yen Kao et al.   HELICOBACTER

Helicobacter pylori perceives the quorum-sensing molecule AI-2 as a chemorepellent via the chemoreceptor TlpB

(2011) B. A. Rader et al.   MICROBIOLOGY-SGM

In Helicobacter pylori auto-inducer-2, but not LuxS/MccAB catalysed reverse transsulphuration, regulates motility through modulation of flagellar gene transcription

(2010) Feifei Shen et al.   BMC MICROBIOLOGY

Detailed In Vivo Analysis of the Role of Helicobacter pylori Fur in Colonization and Disease

(2010) S. Miles et al.   INFECTION AND IMMUNITY

Coordinating assembly of a bacterial macromolecular machine

(2008) Fabienne F. V. Chevance et al.   NATURE REVIEWS MICROBIOLOGY