Differential protein acetylation assists import of excess SOD2 into mitochondria and mediates SOD2 aggregation associated with cardiac hypertrophy in the murine SOD2-tg heart
Differential protein acetylation assists import of excess SOD2 into mitochondria and mediates SOD2 aggregation associated with cardiac hypertrophy in the murine SOD2-tg heart
作者
关键词
SOD2, Mitochondria, Protein acetylation, Protein aggregation, Mitochondrial translocation, Cardiac-specific transgenic mouse
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