The Influenza Virus H5N1 Infection Can Induce ROS Production for Viral Replication and Host Cell Death in A549 Cells Modulated by Human Cu/Zn Superoxide Dismutase (SOD1) Overexpression
出版年份 2016 全文链接
标题
The Influenza Virus H5N1 Infection Can Induce ROS Production for Viral Replication and Host Cell Death in A549 Cells Modulated by Human Cu/Zn Superoxide Dismutase (SOD1) Overexpression
作者
关键词
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出版物
Viruses-Basel
Volume 8, Issue 1, Pages 13
出版商
MDPI AG
发表日期
2016-01-09
DOI
10.3390/v8010013
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Influenza A virus PB1-F2 is involved in regulation of cellular redox state in alveolar epithelial cells
- (2015) Nary Shin et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Alteration of copper–zinc superoxide dismutase 1 expression by influenza A virus is correlated with virus replication
- (2014) Chul-Woong Pyo et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Influenza virus replication in lung epithelial cells depends on redox-sensitive pathways activated by NOX4-derived ROS
- (2014) Donatella Amatore et al. CELLULAR MICROBIOLOGY
- Mitochondria and Metabolic Homeostasis
- (2013) Zhiyong Cheng et al. ANTIOXIDANTS & REDOX SIGNALING
- Cu/Zn superoxide dismutase (SOD1) induction is implicated in the antioxidative and antiviral activity of acetylsalicylic acid in HCV-expressing cells
- (2012) Ana María Rivas-Estilla et al. AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY
- Redox Regulation of the Influenza Hemagglutinin Maturation Process: A New Cell-Mediated Strategy for Anti-Influenza Therapy
- (2011) Rossella Sgarbanti et al. ANTIOXIDANTS & REDOX SIGNALING
- Mitochondrial Ca2+ and ROS take center stage to orchestrate TNF-α–mediated inflammatory responses
- (2011) Laura A. Dada et al. JOURNAL OF CLINICAL INVESTIGATION
- HSV Infection Induces Production of ROS, which Potentiate Signaling from Pattern Recognition Receptors: Role for S-glutathionylation of TRAF3 and 6
- (2011) Regina Gonzalez-Dosal et al. PLoS Pathogens
- Inhibition of Nox2 Oxidase Activity Ameliorates Influenza A Virus-Induced Lung Inflammation
- (2011) Ross Vlahos et al. PLoS Pathogens
- Hepatocyte NAD(P)H oxidases as an endogenous source of reactive oxygen species during hepatitis C virus infection
- (2010) Nabora Soledad Reyes de Mochel et al. HEPATOLOGY
- Hepatitis C virus induces oxidative stress, DNA damage and modulates the DNA repair enzyme NEIL1
- (2010) Sampa Pal et al. JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY
- Highly Pathogenic H5N1 Avian Influenza Virus Induces Extracellular Ca2+ Influx, Leading to Apoptosis in Avian Cells
- (2010) M. Ueda et al. JOURNAL OF VIROLOGY
- Respiratory Syncytial Virus Induces Oxidative Stress by Modulating Antioxidant Enzymes
- (2009) Yashoda M. Hosakote et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- N-acetyl-l-cysteine (NAC) inhibits virus replication and expression of pro-inflammatory molecules in A549 cells infected with highly pathogenic H5N1 influenza A virus
- (2009) Janina Geiler et al. BIOCHEMICAL PHARMACOLOGY
- Bcl-2 Expression and p38MAPK Activity in Cells Infected with Influenza A Virus
- (2009) Lucia Nencioni et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- The Nrf2-Antioxidant Response Element Signaling Pathway and Its Activation by Oxidative Stress
- (2009) Truyen Nguyen et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Identification of Oxidative Stress and Toll-like Receptor 4 Signaling as a Key Pathway of Acute Lung Injury
- (2008) Yumiko Imai et al. CELL
- NADPH oxidases in the vasculature: Molecular features, roles in disease and pharmacological inhibition
- (2008) Stavros Selemidis et al. PHARMACOLOGY & THERAPEUTICS
- H5N1 and 1918 Pandemic Influenza Virus Infection Results in Early and Excessive Infiltration of Macrophages and Neutrophils in the Lungs of Mice
- (2008) Lucy A. Perrone et al. PLoS Pathogens
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