标题
HSP110 promotes colorectal cancer growth through STAT3 activation
作者
关键词
-
出版物
ONCOGENE
Volume 36, Issue 16, Pages 2328-2336
出版商
Springer Nature
发表日期
2016-11-07
DOI
10.1038/onc.2016.403
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- HSPH1 inhibition downregulates Bcl-6 and c-Myc and hampers the growth of human aggressive B-cell non-Hodgkin lymphoma
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- Protective mucosal immunity mediated by epithelial CD1d and IL-10
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- Characterization of an adaptive immune response in microsatellite-instable colorectal cancer
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- Patients With Colorectal Tumors With Microsatellite Instability and Large Deletions in HSP110 T17 Have Improved Response to 5-Fluorouracil–Based Chemotherapy
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- Hsp110 Is aBona FideChaperone Using ATP to Unfold Stable Misfolded Polypeptides and Reciprocally Collaborate with Hsp70 to Solubilize Protein Aggregates
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- Chaperone machines for protein folding, unfolding and disaggregation
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- (2012) Maximilian J. Waldner et al. International Journal of Biological Sciences
- STAT3 interacts directly with Hsp90
- (2012) Earl Prinsloo et al. IUBMB LIFE
- Serological identification of HSP105 as a novel non-Hodgkin lymphoma therapeutic target
- (2011) R. Zappasodi et al. BLOOD
- Expression of a mutant HSP110 sensitizes colorectal cancer cells to chemotherapy and improves disease prognosis
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- Targeting heat shock proteins in cancer
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- gp130-Mediated Stat3 Activation in Enterocytes Regulates Cell Survival and Cell-Cycle Progression during Colitis-Associated Tumorigenesis
- (2009) Julia Bollrath et al. CANCER CELL
- IL-6 and Stat3 Are Required for Survival of Intestinal Epithelial Cells and Development of Colitis-Associated Cancer
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- Hsp105β upregulates hsp70 gene expression through signal transducer and activator of transcription-3
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