Defective proviruses rapidly accumulate during acute HIV-1 infection

Persistent HIV-1 replication maintains the tissue reservoir during therapy

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Clonally expanded CD4+T cells can produce infectious HIV-1 in vivo

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Precise Quantitation of the Latent HIV-1 Reservoir: Implications for Eradication Strategies

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Origin of Rebound Plasma HIV Includes Cells with Identical Proviruses That Are Transcriptionally Active before Stopping of Antiretroviral Therapy

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Broad CTL response is required to clear latent HIV-1 due to dominance of escape mutations

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Interferon-α Subtypes in an Ex Vivo Model of Acute HIV-1 Infection: Expression, Potency and Effector Mechanisms

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Designing and Interpreting Limiting Dilution Assays: General Principles and Applications to the Latent Reservoir for Human Immunodeficiency Virus-1

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Antiretroviral Treatment of Adult HIV Infection

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Proliferation of cells with HIV integrated into cancer genes contributes to persistent infection

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Specific HIV integration sites are linked to clonal expansion and persistence of infected cells

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Replication-Competent Noninduced Proviruses in the Latent Reservoir Increase Barrier to HIV-1 Cure

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An Integrated Overview of HIV-1 Latency

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Emerging strategies to deplete the HIV reservoir

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Antiretroviral Therapy Initiated Within 6 Months of HIV Infection Is Associated With Lower T-Cell Activation and Smaller HIV Reservoir Size

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Highly Precise Measurement of HIV DNA by Droplet Digital PCR

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Comparative Analysis of Measures of Viral Reservoirs in HIV-1 Eradication Studies

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Rapid Quantification of the Latent Reservoir for HIV-1 Using a Viral Outgrowth Assay

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Administration of vorinostat disrupts HIV-1 latency in patients on antiretroviral therapy

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Shock and kill

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Role of retroviral restriction factors in the interferon- -mediated suppression of HIV-1 in vivo

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Immediate antiviral therapy appears to restrict resting CD4+ cell HIV-1 infection without accelerating the decay of latent infection

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Mechanisms and Factors that Influence High Frequency Retroviral Recombination

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Homeostatic Proliferation Fails to Efficiently Reactivate HIV-1 Latently Infected Central Memory CD4+ T Cells

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Induction of a Striking Systemic Cytokine Cascade prior to Peak Viremia in Acute Human Immunodeficiency Virus Type 1 Infection, in Contrast to More Modest and Delayed Responses in Acute Hepatitis B and C Virus Infections

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