Open conformers of HLA-F are high-affinity ligands of the activating NK-cell receptor KIR3DS1
出版年份 2016 全文链接
标题
Open conformers of HLA-F are high-affinity ligands of the activating NK-cell receptor KIR3DS1
作者
关键词
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出版物
NATURE IMMUNOLOGY
Volume 17, Issue 9, Pages 1067-1074
出版商
Springer Nature
发表日期
2016-07-26
DOI
10.1038/ni.3513
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Peptide-Dependent Recognition of HLA-B*57:01 by KIR3DS1
- (2015) Geraldine M. O'Connor et al. JOURNAL OF VIROLOGY
- Efficient Ablation of Genes in Human Hematopoietic Stem and Effector Cells using CRISPR/Cas9
- (2014) Pankaj K. Mandal et al. Cell Stem Cell
- Improved vectors and genome-wide libraries for CRISPR screening
- (2014) Neville E Sanjana et al. NATURE METHODS
- KIR3DS1/L1 and HLA-Bw4-80I are associated with HIV disease progression among HIV typical progressors and long-term nonprogressors
- (2013) Yongjun Jiang et al. BMC INFECTIOUS DISEASES
- Characterization of HLA-F polymorphism in four distinct populations in Mainland China
- (2013) F. H. Pan et al. International Journal of Immunogenetics
- HLA-F and MHC Class I Open Conformers Are Ligands for NK Cell Ig-like Receptors
- (2013) J. P. Goodridge et al. JOURNAL OF IMMUNOLOGY
- The genetic predisposition of natural killer cell to BK virus–associated nephropathy in renal transplant patients
- (2013) Hanna Trydzenskaya et al. KIDNEY INTERNATIONAL
- Natural Killer KIR3DS1 Is Closely Associated with HCV Viral Clearance and Sustained Virological Response in HIV/HCV Patients
- (2013) Antonio Rivero-Juarez et al. PLoS One
- Natural killer cells: walking three paths down memory lane
- (2013) Gundula Min-Oo et al. TRENDS IN IMMUNOLOGY
- HLA Class I Allelic Sequence and Conformation Regulate Leukocyte Ig-Like Receptor Binding
- (2011) D. C. Jones et al. JOURNAL OF IMMUNOLOGY
- Variable NK Cell Receptors Exemplified by Human KIR3DL1/S1
- (2011) P. Parham et al. JOURNAL OF IMMUNOLOGY
- TRIM5 is an innate immune sensor for the retrovirus capsid lattice
- (2011) Thomas Pertel et al. NATURE
- Killer cell immunoglobulin-like receptor 3DL1-mediated recognition of human leukocyte antigen B
- (2011) Julian P. Vivian et al. NATURE
- Copy Number Variation of KIR Genes Influences HIV-1 Control
- (2011) Kimberly Pelak et al. PLOS BIOLOGY
- Natural Killer Cells: From Basic Research to Treatments
- (2011) Eric Vivier et al. Frontiers in Immunology
- Association of the KIR3DS1*013 and KIR3DL1*004 alleles with susceptibility to ankylosing spondylitis
- (2010) Roberto Díaz-Peña et al. ARTHRITIS AND RHEUMATISM
- Donor activating KIR3DS1 is associated with decreased acute GVHD in unrelated allogeneic hematopoietic stem cell transplantation
- (2010) J. M. Venstrom et al. BLOOD
- HLA-F is a surface marker on activated lymphocytes
- (2010) Ni Lee et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- HLA-F Complex without Peptide Binds to MHC Class I Protein in the Open Conformer Form
- (2010) J. P. Goodridge et al. JOURNAL OF IMMUNOLOGY
- Donor KIR3DL1/3DS1 Gene and Recipient Bw4 KIR Ligand as Prognostic Markers for Outcome in Unrelated Hematopoietic Stem Cell Transplantation
- (2009) Katia Gagne et al. BIOLOGY OF BLOOD AND MARROW TRANSPLANTATION
- Dimorphic Motifs in D0 and D1+D2 Domains of Killer Cell Ig-Like Receptor 3DL1 Combine to Form Receptors with High, Moderate, and No Avidity for the Complex of a Peptide Derived from HIV and HLA-A*2402
- (2009) D. Sharma et al. JOURNAL OF IMMUNOLOGY
- Conferral of Enhanced Natural Killer Cell Function by KIR3DS1 in Early Human Immunodeficiency Virus Type 1 Infection
- (2008) B. R. Long et al. JOURNAL OF VIROLOGY
- MHC class I chain-related protein A antibodies and shedding are associated with the progression of multiple myeloma
- (2008) M. Jinushi et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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