Characterisation of neuroprotective efficacy of modified poly-arginine-9 (R9) peptides using a neuronal glutamic acid excitotoxicity model
出版年份 2016 全文链接
标题
Characterisation of neuroprotective efficacy of modified poly-arginine-9 (R9) peptides using a neuronal glutamic acid excitotoxicity model
作者
关键词
Poly-arginine peptides, Arginine-rich peptides, Cortical neurons, Neuroprotection, Glutamate excitotoxicity, Cortical neurons, Cell-penetrating peptides
出版物
MOLECULAR AND CELLULAR BIOCHEMISTRY
Volume 426, Issue 1-2, Pages 75-85
出版商
Springer Nature
发表日期
2016-11-14
DOI
10.1007/s11010-016-2882-z
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Poly-arginine peptides reduce infarct volume in a permanent middle cerebral artery rat stroke model
- (2016) Diego Milani et al. BMC NEUROSCIENCE
- The Neuroprotective Peptide Poly-Arginine-12 (R12) Reduces Cell Surface Levels of NMDA NR2B Receptor Subunit in Cortical Neurons; Investigation into the Involvement of Endocytic Mechanisms
- (2016) Gabriella MacDougall et al. JOURNAL OF MOLECULAR NEUROSCIENCE
- A beacon of hope in stroke therapy—Blockade of pathologically activated cellular events in excitotoxic neuronal death as potential neuroprotective strategies
- (2016) Ashfaqul Hoque et al. PHARMACOLOGY & THERAPEUTICS
- Neuroprotective agents for neonatal hypoxic–ischemic brain injury
- (2015) Qiaofeng Wu et al. DRUG DISCOVERY TODAY
- Inhibition ofN-Methyl-d-aspartate-induced Retinal Neuronal Death by Polyarginine Peptides Is Linked to the Attenuation of Stress-induced Hyperpolarization of the Inner Mitochondrial Membrane Potential
- (2015) John Marshall et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Neuroprotective peptides fused to arginine-rich cell penetrating peptides: Neuroprotective mechanism likely mediated by peptide endocytic properties
- (2015) Bruno P. Meloni et al. PHARMACOLOGY & THERAPEUTICS
- Collapsin Response Mediator Protein 2 (CRMP2) Interacts withN-Methyl-d-aspartate (NMDA) Receptor and Na+/Ca2+Exchanger and Regulates Their Functional Activity
- (2014) Tatiana Brustovetsky et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Glycosaminoglycans in the cellular uptake of drug delivery vectors – Bystanders or active players?
- (2014) Marco E. Favretto et al. JOURNAL OF CONTROLLED RELEASE
- The Deubiquitinating Enzyme USP5 Modulates Neuropathic and Inflammatory Pain by Enhancing Cav3.2 Channel Activity
- (2014) Agustin García-Caballero et al. NEURON
- The Neuroprotective Efficacy of Cell-Penetrating Peptides TAT, Penetratin, Arg-9, and Pep-1 in Glutamic Acid, Kainic Acid, and In Vitro Ischemia Injury Models Using Primary Cortical Neuronal Cultures
- (2013) Bruno P. Meloni et al. CELLULAR AND MOLECULAR NEUROBIOLOGY
- The Mitochondrial-Targeted Compound SS-31 Re-Energizes Ischemic Mitochondria by Interacting with Cardiolipin
- (2013) A. V. Birk et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- Challenging the catechism of therapeutics for chronic neuropathic pain: Targeting CaV2.2 interactions with CRMP2 peptides
- (2013) Polina Feldman et al. NEUROSCIENCE LETTERS
- Neuroprotection against Traumatic Brain Injury by a Peptide Derived from the Collapsin Response Mediator Protein 2 (CRMP2)
- (2011) Joel M. Brittain et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Excitotoxicity-induced endocytosis mediates neuroprotection by TAT-peptide-linked JNK inhibitor
- (2011) Anne Vaslin et al. JOURNAL OF NEUROCHEMISTRY
- DAPK1 Interaction with NMDA Receptor NR2B Subunits Mediates Brain Damage in Stroke
- (2010) Weihong Tu et al. CELL
- Src inhibition reduces NR2B surface expression and synaptic plasticity in the amygdala
- (2010) L. Sinai et al. LEARNING & MEMORY
- Interaction of Postsynaptic Density Protein-95 with NMDA Receptors Influences Excitotoxicity in the Yeast Artificial Chromosome Mouse Model of Huntington's Disease
- (2009) J. Fan et al. JOURNAL OF NEUROSCIENCE
- Mitochondria-Penetrating Peptides
- (2008) Kristin L. Horton et al. CHEMISTRY & BIOLOGY
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More