Extracellular component hyaluronic acid and its receptor Hmmr are required for epicardial EMT during heart regeneration
出版年份 2015 全文链接
标题
Extracellular component hyaluronic acid and its receptor Hmmr are required for epicardial EMT during heart regeneration
作者
关键词
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出版物
CARDIOVASCULAR RESEARCH
Volume 107, Issue 4, Pages 487-498
出版商
Oxford University Press (OUP)
发表日期
2015-07-09
DOI
10.1093/cvr/cvv190
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注意：仅列出部分参考文献，下载原文获取全部文献信息。- Epicardial regeneration is guided by cardiac outflow tract and Hedgehog signalling
- (2015) Jinhu Wang et al. NATURE
- Nrg1 is an injury-induced cardiomyocyte mitogen for the endogenous heart regeneration program in zebrafish
- (2015) Matthew Gemberling et al. eLife
- A Proliferative Burst during Preadolescence Establishes the Final Cardiomyocyte Number
- (2014) Nawazish Naqvi et al. CELL
- Platelet-derived Growth Factor β-Receptor, Transforming Growth Factor β Type I Receptor, and CD44 Protein Modulate Each Other's Signaling and Stability
- (2014) Helena Porsch et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Specific Sizes of Hyaluronan Oligosaccharides Stimulate Fibroblast Migration and Excisional Wound Repair
- (2014) Cornelia Tolg et al. PLoS One
- Notch signaling regulates cardiomyocyte proliferation during zebrafish heart regeneration
- (2014) L. Zhao et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Do Neonatal Mouse Hearts Regenerate following Heart Apex Resection?
- (2014) Ditte Caroline Andersen et al. Stem Cell Reports
- FAK and HAS Inhibition Synergistically Decrease Colon Cancer Cell Viability and Affect Expression of Critical Genes
- (2013) Melissa Heffler et al. Anti-Cancer Agents in Medicinal Chemistry
- Post-transcriptional mechanisms contribute to Etv2 repression during vascular development
- (2013) John C. Moore et al. DEVELOPMENTAL BIOLOGY
- A dynamic spatiotemporal extracellular matrix facilitates epicardial-mediated vertebrate heart regeneration
- (2013) Sarah E. Mercer et al. DEVELOPMENTAL BIOLOGY
- Igf Signaling is Required for Cardiomyocyte Proliferation during Zebrafish Heart Development and Regeneration
- (2013) Ying Huang et al. PLoS One
- Translational profiling of cardiomyocytes identifies an early Jak1/Stat3 injury response required for zebrafish heart regeneration
- (2013) Y. Fang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Hyaluronan: A critical regulator of endothelial-to-mesenchymal transition during cardiac valve formation
- (2013) Anne Karine Lagendijk et al. TRENDS IN CARDIOVASCULAR MEDICINE
- Hyaluronic acid-based hydrogel induces neovascularization and improves cardiac function in a rat model of myocardial infarction
- (2013) Sherif Abdalla et al. Interactive Cardiovascular and Thoracic Surgery
- Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy
- (2012) Sarah Calve et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Migration of cardiomyocytes is essential for heart regeneration in zebrafish
- (2012) J. Itou et al. DEVELOPMENT
- The regenerative capacity of the zebrafish heart is dependent on TGF signaling
- (2012) F. Chablais et al. DEVELOPMENT
- Pan-epicardial lineage tracing reveals that epicardium derived cells give rise to myofibroblasts and perivascular cells during zebrafish heart regeneration
- (2012) Juan Manuel González-Rosa et al. DEVELOPMENTAL BIOLOGY
- In vitro culture of epicardial cells from adult zebrafish heart on a fibrin matrix
- (2012) Jieun Kim et al. Nature Protocols
- Regulation of neonatal and adult mammalian heart regeneration by the miR-15 family
- (2012) E. R. Porrello et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Life-long preservation of the regenerative capacity in the fin and heart in zebrafish
- (2012) J. Itou et al. Biology Open
- tcf21+ epicardial cells adopt non-myocardial fates during zebrafish heart development and regeneration
- (2011) K. Kikuchi et al. DEVELOPMENT
- Retinoic Acid Production by Endocardium and Epicardium Is an Injury Response Essential for Zebrafish Heart Regeneration
- (2011) Kazu Kikuchi et al. DEVELOPMENTAL CELL
- Transient Regenerative Potential of the Neonatal Mouse Heart
- (2011) E. R. Porrello et al. SCIENCE
- Detection of focal adhesion kinase activation at membrane microdomains by fluorescence resonance energy transfer
- (2011) Jihye Seong et al. Nature Communications
- TGFβ2-mediated production of hyaluronan is important for the induction of epicardial cell differentiation and invasion
- (2010) Evisabel A. Craig et al. EXPERIMENTAL CELL RESEARCH
- Primary contribution to zebrafish heart regeneration by gata4 + cardiomyocytes
- (2010) Kazu Kikuchi et al. NATURE
- Zebrafish heart regeneration occurs by cardiomyocyte dedifferentiation and proliferation
- (2010) Chris Jopling et al. NATURE
- PDGF signaling is required for epicardial function and blood vessel formation in regenerating zebrafish hearts
- (2010) J. Kim et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The dual kinase complex FAK-Src as a promising therapeutic target in cancer
- (2010) Victoria Bolós OncoTargets and Therapy
- Early requirement of Hyaluronan for tail regeneration in Xenopus tadpoles
- (2009) E. G. Contreras et al. DEVELOPMENT
- Retro-orbital Injection in Adult Zebrafish
- (2009) Emily K. Pugach et al. Jove-Journal of Visualized Experiments
- Rotation and Asymmetric Development of the Zebrafish Heart Requires Directed Migration of Cardiac Progenitor Cells
- (2008) Kelly A. Smith et al. DEVELOPMENTAL CELL
- Caveolin-1 Up-regulation during Epithelial to Mesenchymal Transition Is Mediated by Focal Adhesion Kinase
- (2008) Kelly M. Bailey et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- FAK regulates cardiomyocyte survival following ischemia/reperfusion
- (2008) Zeenat S. Hakim et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
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