53BP1suppresses epithelial-mesenchymal transition by downregulatingZEB1through microRNA-200b/429 in breast cancer

Bmi1 regulates self-renewal and epithelial to mesenchymal transition in breast cancer cells through Nanog

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Loss of the polycomb protein Mel-18 enhances the epithelial–mesenchymal transition by ZEB1 and ZEB2 expression through the downregulation of miR-205 in breast cancer

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53BP1: pro choice in DNA repair

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53BP1 functions as a tumor suppressor in breast cancer via the inhibition of NF-κB through miR-146a

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Low p53 Binding Protein 1 (53BP1) Expression Is Associated With Increased Local Recurrence in Breast Cancer Patients Treated With Breast-Conserving Surgery and Radiotherapy

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Metadherin enhances the invasiveness of breast cancer cells by inducing epithelial to mesenchymal transition

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p53 regulates epithelial–mesenchymal transition through microRNAs targeting ZEB1 and ZEB2

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p53 regulates epithelial–mesenchymal transition and stem cell properties through modulating miRNAs

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53BP1 loss rescues BRCA1 deficiency and is associated with triple-negative and BRCA-mutated breast cancers

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MTDH Activation by 8q22 Genomic Gain Promotes Chemoresistance and Metastasis of Poor-Prognosis Breast Cancer

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The basics of epithelial-mesenchymal transition

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A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

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The miR-200 Family Inhibits Epithelial-Mesenchymal Transition and Cancer Cell Migration by Direct Targeting of E-cadherin Transcriptional RepressorsZEB1andZEB2

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53BP1 promotes non-homologous end joining of telomeres by increasing chromatin mobility

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The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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