Bok is a genuine multi-BH-domain protein that triggers apoptosis in the absence of Bax and Bak
出版年份 2016 全文链接
标题
Bok is a genuine multi-BH-domain protein that triggers apoptosis in the absence of Bax and Bak
作者
关键词
-
出版物
JOURNAL OF CELL SCIENCE
Volume 129, Issue 11, Pages 2213-2223
出版商
The Company of Biologists
发表日期
2016-04-14
DOI
10.1242/jcs.181727
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- BOK Is a Non-canonical BCL-2 Family Effector of Apoptosis Regulated by ER-Associated Degradation
- (2016) Fabien Llambi et al. CELL
- Is BOK required for apoptosis induced by endoplasmic reticulum stress?
- (2016) Yuniel Fernandez-Marrero et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- BCL-2 family member BOK promotes apoptosis in response to endoplasmic reticulum stress
- (2015) Marcos A. Carpio et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Exploiting selective BCL-2 family inhibitors to dissect cell survival dependencies and define improved strategies for cancer therapy
- (2015) Joel D. Leverson et al. Science Translational Medicine
- Regulating cell death at, on, and in membranes
- (2014) Xiaoke Chi et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
- Many players in BCL-2 family affairs
- (2014) Tudor Moldoveanu et al. TRENDS IN BIOCHEMICAL SCIENCES
- Transcription activator-like effector nucleases (TALENs): A highly efficient and versatile tool for genome editing
- (2013) Ning Sun et al. BIOTECHNOLOGY AND BIOENGINEERING
- Intracellular localization of the BCL-2 family member BOK and functional implications
- (2013) N Echeverry et al. CELL DEATH AND DIFFERENTIATION
- Building blocks of the apoptotic pore: how Bax and Bak are activated and oligomerize during apoptosis
- (2013) D Westphal et al. CELL DEATH AND DIFFERENTIATION
- Control of apoptosis by the BCL-2 protein family: implications for physiology and therapy
- (2013) Peter E. Czabotar et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- Mechanisms of Action of Bcl-2 Family Proteins
- (2013) A. Shamas-Din et al. Cold Spring Harbor Perspectives in Biology
- Targeted therapy of the XIAP/proteasome pathway overcomes TRAIL-resistance in carcinoma by switching apoptosis signaling to a Bax/Bak-independent ‘type I’ mode
- (2013) B Gillissen et al. Cell Death & Disease
- Consequences of the combined loss of BOK and BAK or BOK and BAX
- (2013) F Ke et al. Cell Death & Disease
- BCL-2 family member BOK is widely expressed but its loss has only minimal impact in mice
- (2012) F Ke et al. CELL DEATH AND DIFFERENTIATION
- A Unified Model of Mammalian BCL-2 Protein Family Interactions at the Mitochondria
- (2011) Fabien Llambi et al. MOLECULAR CELL
- Predominant requirement of Bax for apoptosis in HCT116 cells is determined by Mcl-1's inhibitory effect on Bak
- (2011) C Wang et al. ONCOGENE
- BH3 Domains other than Bim and Bid Can Directly Activate Bax/Bak
- (2010) Han Du et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- The landscape of somatic copy-number alteration across human cancers
- (2010) Rameen Beroukhim et al. NATURE
- Inhibition of the ER Ca 2+ pump forces multidrug-resistant cells deficient in Bak and Bax into necrosis
- (2009) Katja Janssen et al. JOURNAL OF CELL SCIENCE
- Puma indirectly activates Bax to cause apoptosis in the absence of Bid or Bim
- (2008) A M Jabbour et al. CELL DEATH AND DIFFERENTIATION
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started