期刊
IMMUNITY
卷 55, 期 4, 页码 592-605出版社
CELL PRESS
DOI: 10.1016/j.immuni.2022.03.016
关键词
-
类别
资金
- Abby and Howard P. Milstein Program in Chemical Biology and Translational Medicine
- Randolph Hearst Trust
Nonresolving inflammation contributes to various diseases, including COVID-19. Our understanding of inflammation is constantly evolving, viewing it as an interactive component of a homeostatic network with the endocrine and nervous systems. This review highlights emerging insights into inflammatory memory, inflammatory aging, inflammatory cell death, inflammatory DNA, inflammation-regulating cells and metabolites, approaches to resolving or modulating inflammation, and inflammatory inequity.
Nonresolving inflammation contributes to many diseases, including COVID-19 in its fatal and long forms. Our understanding of inflammation is rapidly evolving. Like the immune system of which it is a part, inflammation can now be seen as an interactive component of a homeostatic network with the endocrine and nervous systems. This review samples emerging insights regarding inflammatory memory, inflammatory aging, inflammatory cell death, inflammatory DNA, inflammation-regulating cells and metabolites, approaches to resolving or modulating inflammation, and inflammatory inequity.
作者
我是这篇论文的作者
点击您的名字以认领此论文并将其添加到您的个人资料中。
推荐
暂无数据