PM2.5 induces the distant metastasis of lung adenocarcinoma via promoting the stem cell properties of cancer cells
出版年份 2021 全文链接
标题
PM2.5 induces the distant metastasis of lung adenocarcinoma via promoting the stem cell properties of cancer cells
作者
关键词
PM, 2.5, Metastasis, Lung cancer, PDX model, Cancer stem cell, miRNA
出版物
ENVIRONMENTAL POLLUTION
Volume 296, Issue -, Pages 118718
出版商
Elsevier BV
发表日期
2021-12-20
DOI
10.1016/j.envpol.2021.118718
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Long-term PM2.5 exposure increases the risk of non-small cell lung cancer (NSCLC) progression by enhancing interleukin-17a (IL-17a)-regulated proliferation and metastasis
- (2020) Xie Chao et al. Aging-US
- PM2.5 induces EMT and promotes CSC properties by activating Notch pathway in vivo and vitro
- (2019) Yunxia Wang et al. ECOTOXICOLOGY AND ENVIRONMENTAL SAFETY
- Spatio-Temporal Variation Characteristics of PM2.5 in the Beijing–Tianjin–Hebei Region, China, from 2013 to 2018
- (2019) Lili Wang et al. International Journal of Environmental Research and Public Health
- PM2.5, Fine Particulate Matter: A Novel Player in the Epithelial-Mesenchymal Transition?
- (2019) Zihan Xu et al. Frontiers in Physiology
- Temporal-spatial characteristics and source apportionment of PM 2.5 as well as its associated chemical species in the Beijing-Tianjin-Hebei region of China
- (2018) Jiajia Gao et al. ENVIRONMENTAL POLLUTION
- In Vitro and In Vivo Experimental Studies of PM2.5 on Disease Progression
- (2018) Ching-Chang Cho et al. International Journal of Environmental Research and Public Health
- Spatiotemporal patterns of recent PM2.5 concentrations over typical urban agglomerations in China
- (2018) Yang Shen et al. SCIENCE OF THE TOTAL ENVIRONMENT
- Spatial and temporal distribution, chemical characteristics, and sources of ambient particulate matter in the Beijing-Tianjin-Hebei region
- (2018) Hong Xu et al. SCIENCE OF THE TOTAL ENVIRONMENT
- Exposure to PM2.5 induces aberrant activation of NF-κB in human airway epithelial cells by downregulating miR-331 expression
- (2017) Lei Song et al. ENVIRONMENTAL TOXICOLOGY AND PHARMACOLOGY
- Characterization and expression of lin-28a involved in lin28/let-7signal pathway during early development of P. olivaceus
- (2017) Yuanshuai Fu et al. FISH PHYSIOLOGY AND BIOCHEMISTRY
- Review of receptor-based source apportionment research of fine particulate matter and its challenges in China
- (2017) Yanjun Zhang et al. SCIENCE OF THE TOTAL ENVIRONMENT
- EMT: 2016
- (2016) M. Angela Nieto et al. CELL
- Arsenic and chromium topsoil levels and cancer mortality in Spain
- (2016) Olivier Núñez et al. ENVIRONMENTAL SCIENCE AND POLLUTION RESEARCH
- Spatiotemporal patterns of remotely sensed PM 2.5 concentration in China from 1999 to 2011
- (2016) Jian Peng et al. REMOTE SENSING OF ENVIRONMENT
- Heavy haze episodes in Beijing during January 2013: Inorganic ion chemistry and source analysis using highly time-resolved measurements from an urban site
- (2016) Bin Han et al. SCIENCE OF THE TOTAL ENVIRONMENT
- PRMT1 Is a Novel Regulator of Epithelial-Mesenchymal-Transition in Non-small Cell Lung Cancer
- (2015) Sreedevi Avasarala et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Lung cancer incidence and ambient air pollution in China: a spatial age–period cohort study 1990–2009
- (2015) Yuming Guo et al. LANCET
- High secondary aerosol contribution to particulate pollution during haze events in China
- (2014) Ru-Jin Huang et al. NATURE
- Characterization and Source Apportionment of PM2.5 in an Urban Environment in Beijing
- (2013) Lingda Yu et al. Aerosol and Air Quality Research
- Air pollution and lung cancer incidence in 17 European cohorts: prospective analyses from the European Study of Cohorts for Air Pollution Effects (ESCAPE)
- (2013) Ole Raaschou-Nielsen et al. LANCET ONCOLOGY
- Potential health risks of heavy metals in cultivated topsoil and grain, including correlations with human primary liver, lung and gastric cancer, in Anhui province, Eastern China
- (2013) Qihong Zhao et al. SCIENCE OF THE TOTAL ENVIRONMENT
- TGF-β signaling and epithelial–mesenchymal transition in cancer progression
- (2012) Yoko Katsuno et al. CURRENT OPINION IN ONCOLOGY
- Chronic Exposure to Fine Particles and Mortality: An Extended Follow-up of the Harvard Six Cities Study from 1974 to 2009
- (2012) Johanna Lepeule et al. ENVIRONMENTAL HEALTH PERSPECTIVES
- Epithelial-Mesenchymal Transition: General Principles and Pathological Relevance with Special Emphasis on the Role of Matrix Metalloproteinases
- (2012) P. Nistico et al. Cold Spring Harbor Perspectives in Biology
- Long-term Ambient Fine Particulate Matter Air Pollution and Lung Cancer in a Large Cohort of Never-Smokers
- (2011) Michelle C. Turner et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
- Downregulation of transcription factor SOX2 in cancer stem cells suppresses growth and metastasis of lung cancer
- (2011) R Xiang et al. BRITISH JOURNAL OF CANCER
- MicroRNA-30a inhibits epithelial-to-mesenchymal transition by targeting Snai1 and is downregulated in non-small cell lung cancer
- (2011) Regalla Kumarswamy et al. INTERNATIONAL JOURNAL OF CANCER
- Coexpression of Oct4 and Nanog Enhances Malignancy in Lung Adenocarcinoma by Inducing Cancer Stem Cell-Like Properties and Epithelial-Mesenchymal Transdifferentiation
- (2010) S.-H. Chiou et al. CANCER RESEARCH
- Core epithelial-to-mesenchymal transition interactome gene-expression signature is associated with claudin-low and metaplastic breast cancer subtypes
- (2010) J. H. Taube et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells
- (2008) Ulrike Burk et al. EMBO REPORTS
- The miR-200 Family Inhibits Epithelial-Mesenchymal Transition and Cancer Cell Migration by Direct Targeting of E-cadherin Transcriptional RepressorsZEB1andZEB2
- (2008) Manav Korpal et al. JOURNAL OF BIOLOGICAL CHEMISTRY
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started