期刊
INSECT BIOCHEMISTRY AND MOLECULAR BIOLOGY
卷 73, 期 -, 页码 55-61出版社
PERGAMON-ELSEVIER SCIENCE LTD
DOI: 10.1016/j.ibmb.2016.04.005
关键词
Brown planthopper; Wing; Polyphenism; JNK signaling; Jun; Fos; FOXO
资金
- National Natural Science Foundation of China [31471771]
- Natural Science Foundation of Zhejiang Province [Y14C140007]
- National Basic Research Program (973) of China [2010CB126200]
Wing polyphenism is considered to be an adaptive trade-off between migration (long winged forms) and reproduction (short winged forms), determined by various environmental conditions. The c-Jun NH2-terminal kinase (JNK) is crucial for the regulation of the activity of a number of transcription factors, and is activated under stress and environmental fluctuations where it functions in maintaining cell viability and proliferation. We used RNA interference and a pharmacological inhibitor of JNK to test the role of JNK signaling in regulating the wing dimorphism of the brown planthopper, Nilaparvata lugens. Silencing NlJNK increased the proportion of short winged female adults, reminiscent of the effect of silencing inhibitory components of the insulin-signaling pathway, such as NlAkt. However, silencing of the JNK-activated transcription factors NlJun and NlFos did not change the wing form ratio significantly, indicating that NlJNK may not act through NlJun and NlFos in mediating this process. In summary, JNK signaling may play a role in determining wing polymorphism in N. lugens females. (C) 2016 Elsevier Ltd. All rights reserved.
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