Up-regulation of TDAG51 is a dependent factor of LPS-induced RAW264.7 macrophages proliferation and cell cycle progression

Involvement of monocytes/macrophages as key factors in the development and progression of cardiovascular diseases

(2014) María Fernández-Velasco et al.   BIOCHEMICAL JOURNAL

The involvement of the monocytes/macrophages in chronic inflammation associated with atherosclerosis

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Deficiency of TDAG51 Protects Against Atherosclerosis by Modulating Apoptosis, Cholesterol Efflux, and Peroxiredoxin‐1 Expression

(2013) Gazi S. Hossain et al.   Journal of the American Heart Association

Downregulation of miR-27a* and miR-532-5p and Upregulation of miR-146a and miR-155 in LPS-induced RAW264.7 Macrophage Cells

(2012) Ying Cheng et al.   INFLAMMATION

α-Mangostin Induces Apoptosis and Suppresses Differentiation of 3T3-L1 Cells via Inhibiting Fatty Acid Synthase

(2012) Xiaofang Quan et al.   PLoS One

siRNA targeting mCD14 inhibits TNF-α, MIP-2, and IL-6 secretion and NO production from LPS-induced RAW264.7 cells

(2011) Ming Lei et al.   APPLIED MICROBIOLOGY AND BIOTECHNOLOGY

Type II interleukin-1 receptor expression is reduced in monocytes/macrophages and atherosclerotic lesions

(2011) Jordi Pou et al.   BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR AND CELL BIOLOGY OF LIPIDS

PHLDA1 Expression Marks the Putative Epithelial Stem Cells and Contributes to Intestinal Tumorigenesis

(2011) A. Sakthianandeswaren et al.   CANCER RESEARCH

TDAG51 is an ERK signaling target that opposes ERK-mediated HME16C mammary epithelial cell transformation

(2008) Michael D Oberst et al.   BMC CANCER

Phenotypic non-equivalence of murine (monocyte-) macrophage cells in biomaterial and inflammatory models

(2008) Lisa M. Chamberlain et al.   JOURNAL OF BIOMEDICAL MATERIALS RESEARCH PART A