期刊
RICE
卷 14, 期 1, 页码 -出版社
SPRINGER
DOI: 10.1186/s12284-021-00493-w
关键词
Rice; Senescence; IMBR; Heat tolerance; Hydrogen peroxide
类别
资金
- Central Public-interest Scientific Institution Basal Research Fund of China National Rice Research Institute [2017RG002-2]
The study identified a premature senescence leaf 50 (psl50) mutant in rice with a higher susceptibility to heat stress, over-accumulation of hydrogen peroxide (H2O2), and increased cell death. The causal gene PSL50 was found to negatively regulate heat-induced premature leaf senescence in rice by modulating H2O2 signaling.
Background Heat stress is a major environmental factor that could induce premature leaf senescence in plants. So far, a few rice premature senescent leaf mutants have been reported to involve in heat tolerance. Findings We identified a premature senescence leaf 50 (psl50) mutant that exhibited a higher heat susceptibility with decreased survival rate, over-accumulated hydrogen peroxide (H2O2) content and increased cell death under heat stress compared with the wild-type. The causal gene PREMATURE SENESCENCE LEAF 50 (PSL50) was isolated by using initial map-based resequencing (IMBR) approach, and we found that PSL50 promoted heat tolerance probably by acting as a modulator of H2O2 signaling in response to heat stress in rice (Oryza sativa L.). Conclusions PSL50 negatively regulates heat-induced premature leaf senescence in rice.
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