Lipocalin 2 increases after high‐intensity exercise in humans and influences muscle gene expression and differentiation in mice
出版年份 2021 全文链接
标题
Lipocalin 2 increases after high‐intensity exercise in humans and influences muscle gene expression and differentiation in mice
作者
关键词
-
出版物
JOURNAL OF CELLULAR PHYSIOLOGY
Volume -, Issue -, Pages -
出版商
Wiley
发表日期
2021-07-05
DOI
10.1002/jcp.30501
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Myositis in Lewis rats induced by the superantigen Staphylococcal enterotoxin A
- (2019) Alexander Emmer et al. MOLECULAR BIOLOGY REPORTS
- Non-immunogenic utrophin gene therapy for the treatment of muscular dystrophy animal models
- (2019) Yafeng Song et al. NATURE MEDICINE
- A Complex Role for Lipocalin 2 in Bone Metabolism: Global Ablation in Mice Induces Osteopenia Caused by an Altered Energy Metabolism
- (2018) Mattia Capulli et al. JOURNAL OF BONE AND MINERAL RESEARCH
- Loss of the Adipokine Lipocalin-2 Impairs Satellite Cell Activation and Skeletal Muscle Regeneration
- (2018) Irena A Rebalka et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Lipocalin-2 (NGAL) Attenuates Autophagy to Exacerbate Cardiac Apoptosis Induced by Myocardial Ischemia
- (2017) Hye Kyoung Sung et al. JOURNAL OF CELLULAR PHYSIOLOGY
- MC4R-dependent suppression of appetite by bone-derived lipocalin 2
- (2017) Ioanna Mosialou et al. NATURE
- Experimental and Human Evidence for Lipocalin‐2 (Neutrophil Gelatinase‐Associated Lipocalin [NGAL]) in the Development of Cardiac Hypertrophy and heart failure
- (2017) Francine Z. Marques et al. Journal of the American Heart Association
- Microgravity-Induced Transcriptome Adaptation in Mouse Paraspinal longissimus dorsi Muscle Highlights Insulin Resistance-Linked Genes
- (2017) Guido Gambara et al. Frontiers in Physiology
- Wnt/β-catenin signaling via Axin2 is required for myogenesis and, together with YAP/Taz and Tead1, active in IIa/IIx muscle fibers
- (2016) Danyil Huraskin et al. DEVELOPMENT
- Interleukin-1β, lipocalin 2 and nitric oxide synthase 2 are mechano-responsive mediators of mouse and human endothelial cell-osteoblast crosstalk
- (2016) Vimal Veeriah et al. Scientific Reports
- The potential of lipocalin-2/NGAL as biomarker for inflammatory and metabolic diseases
- (2015) Vanessa Abella et al. BIOMARKERS
- Lipocalin 2: A New Mechanoresponding Gene Regulating Bone Homeostasis
- (2015) Nadia Rucci et al. JOURNAL OF BONE AND MINERAL RESEARCH
- Altered bone development and turnover in transgenic mice over-expressing Lipocalin-2 in bone
- (2013) Delfina Costa et al. JOURNAL OF CELLULAR PHYSIOLOGY
- Therapy for Duchenne muscular dystrophy: renewed optimism from genetic approaches
- (2013) Rebecca J. Fairclough et al. NATURE REVIEWS GENETICS
- Lipocalin-2 increases fat oxidation in vitro and is correlated with energy expenditure in normal weight but not obese women
- (2013) Chad M. Paton et al. Obesity
- Lipocalin-2 (LCN-2) is a Major Acute Phase Protein in a Rat and Mouse Model of Sterile Abscess
- (2012) Sadaf Sultan et al. SHOCK
- Lipocalin-2 Induces Cardiomyocyte Apoptosis by Increasing Intracellular Iron Accumulation
- (2011) Guoxiong Xu et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Lipocalin-2/Neutrophil Gelatinase-B Associated Lipocalin Is Strongly Induced in Hearts of Rats With Autoimmune Myocarditis and in Human Myocarditis
- (2010) Limin Ding et al. CIRCULATION JOURNAL
- Myoblast sensitivity and fibroblast insensitivity to osteogenic conversion by BMP-2 correlates with the expression of Bmpr-1a
- (2009) Renjing Liu et al. BMC MUSCULOSKELETAL DISORDERS
- Global transcriptome analysis in mouse calvarial osteoblasts highlights sets of genes regulated by modeled microgravity and identifies a “mechanoresponsive osteoblast gene signature”
- (2009) Mattia Capulli et al. JOURNAL OF CELLULAR BIOCHEMISTRY
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