Cardiac abnormalities after induction of endoplasmic reticulum stress are associated with mitochondrial dysfunction and connexin43 expression
出版年份 2021 全文链接
标题
Cardiac abnormalities after induction of endoplasmic reticulum stress are associated with mitochondrial dysfunction and connexin43 expression
作者
关键词
-
出版物
CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY
Volume 48, Issue 10, Pages 1371-1381
出版商
Wiley
发表日期
2021-06-17
DOI
10.1111/1440-1681.13541
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Mitochondrial Cx43, an important component of cardiac preconditioning
- (2018) Antonio Rodríguez-Sinovas et al. BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES
- Over-expression of calpastatin attenuates myocardial injury following myocardial infarction by inhibiting endoplasmic reticulum stress
- (2018) Shuai Li et al. Journal of Thoracic Disease
- Alogliptin, a Dipeptidyl Peptidase‐4 Inhibitor, Alleviates Atrial Remodeling and Improves Mitochondrial Function and Biogenesis in Diabetic Rabbits
- (2017) Xiaowei Zhang et al. Journal of the American Heart Association
- Unfolded protein response plays a critical role in heart damage after myocardial ischemia/reperfusion in rats
- (2017) Chengcheng Zhang et al. PLoS One
- Uric acid enhances PKC-dependent eNOS phosphorylation and mediates cellular ER stress: A mechanism for uric acid-induced endothelial dysfunction
- (2016) PENG LI et al. INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE
- Chemical chaperone 4-phenylbutyric acid protects H9c2 cardiomyocytes from ischemia/reperfusion injury by attenuating endoplasmic reticulum stress-induced apoptosis
- (2016) LIAN JIAN et al. Molecular Medicine Reports
- PGC-1α controls mitochondrial biogenesis and dynamics in lead-induced neurotoxicity
- (2015) Aleksandra Dabrowska et al. Aging-US
- Cell death induced by endoplasmic reticulum stress
- (2015) Raffaella Iurlaro et al. FEBS Journal
- The therapeutic effects of 4-phenylbutyric acid in maintaining proteostasis
- (2015) P.S. Kolb et al. INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY
- A novel endoplasmic reticulum stress-induced apoptosis model using tunicamycin in primary cultured neonatal rat cardiomyocytes
- (2015) MINGZHI SHEN et al. Molecular Medicine Reports
- Structure and function of ER membrane contact sites with other organelles
- (2015) Melissa J. Phillips et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- UPR, autophagy, and mitochondria crosstalk underlies the ER stress response
- (2015) Daniela Senft et al. TRENDS IN BIOCHEMICAL SCIENCES
- Evidence that Chemical Chaperone 4-Phenylbutyric Acid Binds to Human Serum Albumin at Fatty Acid Binding Sites
- (2015) Debasish Roy et al. PLoS One
- Unfolded Protein Response Regulates Cardiac Sodium Current in Systolic Human Heart Failure
- (2013) Ge Gao et al. Circulation-Arrhythmia and Electrophysiology
- The China Patient-Centered Evaluative Assessment of Cardiac Events (China PEACE) Retrospective Study of Acute Myocardial Infarction: Study Design
- (2013) Kumar Dharmarajan et al.
- Functional and morphological impact of ER stress on mitochondria
- (2013) Kayleen Vannuvel et al. JOURNAL OF CELLULAR PHYSIOLOGY
- Ca2+/Calmodulin-dependent Protein Kinase II (CaMKII) Regulates Cardiac Sodium Channel NaV1.5 Gating by Multiple Phosphorylation Sites
- (2012) Nicole M. Ashpole et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- In vivo cellular adaptation to ER stress: survival strategies with double-edged consequences
- (2010) K. Y. Tsang et al. JOURNAL OF CELL SCIENCE
- ER stress in cardiovascular disease
- (2009) Tetsuo Minamino et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- TZDs reduce mitochondrial ROS production and enhance mitochondrial biogenesis
- (2008) Kazuo Fujisawa et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Cell death and endoplasmic reticulum stress: disease relevance and therapeutic opportunities
- (2008) Inki Kim et al. NATURE REVIEWS DRUG DISCOVERY
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started