Short-term but not long-term high fat diet feeding protects against pressure overload-induced heart failure through activation of mitophagy

Increasing Fatty Acid Oxidation Prevents High Fat Diet Induced Cardiomyopathy through Regulating Parkin Mediated Mitophagy

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Mitophagy Is Essential for Maintaining Cardiac Function During High Fat Diet-Induced Diabetic Cardiomyopathy

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Enhancing fatty acid utilization ameliorates mitochondrial fragmentation and cardiac dysfunction via rebalancing optic atrophy 1 processing in the failing heart

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Short-term high-fat diet compromises myocardial function: a radial strain rate imaging study

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Drp1-Dependent Mitochondrial Autophagy Plays a Protective Role Against Pressure Overload–Induced Mitochondrial Dysfunction and Heart FailureCLINICAL PERSPECTIVE

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Self-renewal of a purified Tie2+ hematopoietic stem cell population relies on mitochondrial clearance

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Good and bad consequences of altered fatty acid metabolism in heart failure: evidence from mouse models

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Molecular Mechanisms of Mitochondrial Autophagy/Mitophagy in the Heart

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Fatty Acid Signaling: The New Function of Intracellular Lipases

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Imbalanced OPA1 processing and mitochondrial fragmentation cause heart failure in mice

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Mitochondrial fatty acid oxidation alterations in heart failure, ischaemic heart disease and diabetic cardiomyopathy

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Carnitine Palmitoyltransferase-1b Deficiency Aggravates Pressure Overload–Induced Cardiac Hypertrophy Caused by Lipotoxicity

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Cardiac-Specific Deletion of Acetyl CoA Carboxylase 2 Prevents Metabolic Remodeling During Pressure-Overload Hypertrophy

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A short duration of high-fat diet induces insulin resistance and predisposes to adverse left ventricular remodeling after pressure overload

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Low-Carbohydrate/High-Fat Diet Attenuates Pressure Overload–Induced Ventricular Remodeling and Dysfunction

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