Rapid fall in circulating non‐classical monocytes in ST elevation myocardial infarction patients correlates with cardiac injury

Revising CX3CR1 Expression on Murine Classical and Non-classical Monocytes

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Enhanced monocyte recruitment and delayed alternative macrophage polarisation accompanies impaired repair following myocardial infarction in C57 BL /6 compared to BALB /c mice

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PD-L1 expression on nonclassical monocytes reveals their origin and immunoregulatory function

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Monocyte Subsets Coregulate Inflammatory Responses by Integrated Signaling through TNF and IL-6 at the Endothelial Cell Interface

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Cardiosphere-Derived Cells Require Endoglin for Paracrine-Mediated Angiogenesis

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The Kinetics of Circulating Monocyte Subsets and Monocyte-Platelet Aggregates in the Acute Phase of ST-Elevation Myocardial Infarction

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Role and analysis of monocyte subsets in cardiovascular disease

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Inflammation in cardiac injury, repair and regeneration

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Acute myocardial infarction activates distinct inflammation and proliferation pathways in circulating monocytes, prior to recruitment, and identified through conserved transcriptional responses in mice and humans

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T lymphocytes and fractalkine contribute to myocardial ischemia/reperfusion injury in patients

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Endoscopic Time-Lapse Imaging of Immune Cells in Infarcted Mouse Hearts

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Temporal dynamics of cardiac immune cell accumulation following acute myocardial infarction

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Rapid monocyte kinetics in acute myocardial infarction are sustained by extramedullary monocytopoiesis

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Final infarct size measured by cardiovascular magnetic resonance in patients with ST elevation myocardial infarction predicts long-term clinical outcome: an observational study

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The CD14++CD16+ monocyte subset and monocyte-platelet interactions in patients with ST-elevation myocardial infarction

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Comparison of gene expression profiles between human and mouse monocyte subsets

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Impact of Heterogeneity of Human Peripheral Blood Monocyte Subsets on Myocardial Salvage in Patients With Primary Acute Myocardial Infarction

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Identification of Splenic Reservoir Monocytes and Their Deployment to Inflammatory Sites

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