期刊
NATURE NEUROSCIENCE
卷 24, 期 2, 页码 157-159出版社
NATURE PORTFOLIO
DOI: 10.1038/s41593-020-00777-2
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This study shows that local proliferation of CNS fibroblasts drives fibrotic scar formation in response to circulating inflammatory cell infiltration in a model of multiple sclerosis, which may limit repair.
Fibrotic scarring after inflammation is well-characterized in peripheral tissues, but its role in the CNS is less clear. A new study shows that local proliferation of CNS fibroblasts drives fibrotic scar formation in response to circulating inflammatory cell infiltration in a model of multiple sclerosis, which may limit repair.
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