Clonal distribution of BCR-ABL1 mutations and splice isoforms by single-molecule long-read RNA sequencing
出版年份 2015 全文链接
标题
Clonal distribution of BCR-ABL1 mutations and splice isoforms by single-molecule long-read RNA sequencing
作者
关键词
Imatinib, Dasatinib, Nilotinib, Chronic Myeloid Leukemia Patient, T315I Mutation
出版物
BMC CANCER
Volume 15, Issue 1, Pages -
出版商
Springer Nature
发表日期
2015-02-11
DOI
10.1186/s12885-015-1046-y
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Many BCR-ABL1 compound mutations reported in chronic myeloid leukemia patients may actually be artifacts due to PCR-mediated recombination
- (2014) W. T. Parker et al. BLOOD
- BCR-ABL1 Compound Mutations Combining Key Kinase Domain Positions Confer Clinical Resistance to Ponatinib in Ph Chromosome-Positive Leukemia
- (2014) Matthew S. Zabriskie et al. CANCER CELL
- Molecular dynamics reveal BCR-ABL1 polymutants as a unique mechanism of resistance to PAN-BCR-ABL1 kinase inhibitor therapy
- (2014) Don L. Gibbons et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- European LeukemiaNet recommendations for the management of chronic myeloid leukemia: 2013
- (2013) M. Baccarani et al. BLOOD
- Unraveling the complexity of tyrosine kinase inhibitor-resistant populations by ultra-deep sequencing of the BCR-ABL kinase domain
- (2013) S. Soverini et al. BLOOD
- Rapid identification of compound mutations in patients with Philadelphia-positive leukaemias by long-range next generation sequencing
- (2013) R. Kastner et al. EUROPEAN JOURNAL OF CANCER
- BCR-ABL1 compound mutations in tyrosine kinase inhibitor-resistant CML: frequency and clonal relationships
- (2012) J. S. Khorashad et al. BLOOD
- The BCR-ABL35INS insertion/truncation mutant is kinase-inactive and does not contribute to tyrosine kinase inhibitor resistance in chronic myeloid leukemia
- (2011) T. O'Hare et al. BLOOD
- Three novel alternative splicing mutations in BCR-ABL1 detected in CML patients with resistance to kinase inhibitors
- (2011) W. MA et al. International Journal of Laboratory Hematology
- BCR-ABL isoforms associated with intrinsic or acquired resistance to imatinib: more heterogeneous than just ABL kinase domain point mutations?
- (2011) Franz X. Gruber et al. MEDICAL ONCOLOGY
- BCR-ABL SH3-SH2 domain mutations in chronic myeloid leukemia patients on imatinib
- (2010) D. W. Sherbenou et al. BLOOD
- Exon 7 Deletion in the bcr-abl Gene Is Frequent in Chronic Myeloid Leukemia Patients and Is Not Correlated with Resistance against Imatinib
- (2010) J.-B. Gaillard et al. MOLECULAR CANCER THERAPEUTICS
- An Intron-Derived Insertion/Truncation Mutation in the BCR-ABL Kinase Domain in Chronic Myeloid Leukemia Patients Undergoing Kinase Inhibitor Therapy
- (2008) Jennifer Laudadio et al. JOURNAL OF MOLECULAR DIAGNOSTICS
- Characterization of BCR-ABL deletion mutants from patients with chronic myeloid leukemia
- (2008) D W Sherbenou et al. LEUKEMIA
- BCR-ABL alternative splicing as a common mechanism for imatinib resistance: evidence from molecular dynamics simulations
- (2008) T.-S. Lee et al. MOLECULAR CANCER THERAPEUTICS
- Real-Time DNA Sequencing from Single Polymerase Molecules
- (2008) J. Eid et al. SCIENCE
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