Histone 2A Family Member J Drives Mesenchymal Transition and Temozolomide Resistance in Glioblastoma Multiforme

The roles of DNA, RNA and histone methylation in ageing and cancer

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Longitudinal molecular trajectories of diffuse glioma in adults

(2019) Floris P. Barthel et al.   NATURE

Inhibition of NF-κB results in anti-glioma activity and reduces temozolomide-induced chemoresistance by down-regulating MGMT gene expression

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Patterns of care and outcomes of chemoradiation versus radiation alone for MGMT promoter unmethylated glioblastoma

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H2AFX and MDC1 promote maintenance of genomic integrity in male germ cells

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Post-Translational Modifications of H2A Histone Variants and Their Role in Cancer

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Targeting the mesenchymal subtype in glioblastoma and other cancers via inhibition of diacylglycerol kinase alpha

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Evolution of histone H3: emergence of variants and conservation of post-translational modification sites1This article is part of Special Issue entitled Asilomar Chromatin and has undergone the Journal’s usual peer review process.

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The 2016 World Health Organization Classification of Tumors of the Central Nervous System: a summary

(2016) David N. Louis et al.   ACTA NEUROPATHOLOGICA

IDH mutation and MGMT promoter methylation are associated with the pseudoprogression and improved prognosis of glioblastoma multiforme patients who have undergone concurrent and adjuvant temozolomide-based chemoradiotherapy

(2016) Hailong Li et al.   CLINICAL NEUROLOGY AND NEUROSURGERY

Lateral Thinking: How Histone Modifications Regulate Gene Expression

(2016) Moyra Lawrence et al.   TRENDS IN GENETICS

COL3A1 and SNAP91: novel glioblastoma markers with diagnostic and prognostic value

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Clonal Variation in Drug and Radiation Response among Glioma-Initiating Cells Is Linked to Proneural-Mesenchymal Transition

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Correlating chemical sensitivity and basal gene expression reveals mechanism of action

(2015) Matthew G Rees et al.   Nature Chemical Biology

Harnessing Connectivity in a Large-Scale Small-Molecule Sensitivity Dataset

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IDH1 mutation and MGMT methylation status predict survival in patients with anaplastic astrocytoma treated with temozolomide-based chemoradiotherapy

(2014) Giuseppe Minniti et al.   JOURNAL OF NEURO-ONCOLOGY

Significance ofCASP8AP2andH2AFZexpression in survival and risk of relapse in children with acute lymphoblastic leukemia

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Mesenchymal Differentiation Mediated by NF-κB Promotes Radiation Resistance in Glioblastoma

(2013) Krishna P.L. Bhat et al.   CANCER CELL

IDH1/2 mutations target a key hallmark of cancer by deregulating cellular metabolism in glioma

(2013) Chunzhi Zhang et al.   NEURO-ONCOLOGY

MGMT repletion after treatment of glioblastoma cells with temozolomide and O6-benzylguanine implicates NFκB and mutant p53

(2013) Panagiotis J Vlachostergios et al.   NEUROLOGICAL RESEARCH

SurvExpress: An Online Biomarker Validation Tool and Database for Cancer Gene Expression Data Using Survival Analysis

(2013) Raul Aguirre-Gamboa et al.   PLoS One

Long non-coding RNA expression profiles predict clinical phenotypes in glioma

(2012) Xiaoqin Zhang et al.   NEUROBIOLOGY OF DISEASE

Immunohistochemical Analysis of Histone H3 Modifications in Germ Cells during Mouse Spermatogenesis

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H2AFX Polymorphisms Are Associated with Decreased Risk of Diffuse Large B Cell Lymphoma in Koreans

(2011) Xue Mei Jin et al.   DNA AND CELL BIOLOGY

Possible association between genetic variants in the H2AFX promoter region and risk of adult glioma in a Chinese Han population

(2011) Weiwei Fan et al.   JOURNAL OF NEURO-ONCOLOGY

Integrated Genomic Analysis Identifies Clinically Relevant Subtypes of Glioblastoma Characterized by Abnormalities in PDGFRA, IDH1, EGFR, and NF1

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All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2

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NFKBIADeletion in Glioblastomas

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The transcriptional network for mesenchymal transformation of brain tumours

(2009) Maria Stella Carro et al.   NATURE

IDH1 mutations in low-grade astrocytomas predict survival but not response to temozolomide

(2009) H. J. Dubbink et al.   NEUROLOGY

MGMTPromoter Methylation Status Can Predict the Incidence and Outcome of Pseudoprogression After Concomitant Radiochemotherapy in Newly Diagnosed Glioblastoma Patients

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