期刊
BMC CELL BIOLOGY
卷 17, 期 -, 页码 -出版社
BIOMED CENTRAL LTD
DOI: 10.1186/s12860-016-0090-z
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资金
- NIDCD NIH HHS [R29 DC003186, R01 DC003186, R56 DC003186, R01 DC011099] Funding Source: Medline
- NINDS NIH HHS [R21 NS085772, R21 NS055726] Funding Source: Medline
Gap junctions mediate electrical synaptic transmission between neurons. While the actions of neurotransmitter modulators on the conductance of gap junctions have been extensively documented, increasing evidence indicates they can also be influenced by the ongoing activity of neural networks, in most cases via local interactions with nearby glutamatergic synapses. We review here early evidence for the existence of activity-dependent regulatory mechanisms as well recent examples reported in mammalian brain. The ubiquitous distribution of both neuronal connexins and the molecules involved suggest this phenomenon is widespread and represents a property of electrical transmission in general.
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