eIF2α-CHOP-BCl-2/JNK and IRE1α-XBP1/JNK signaling promote apoptosis and inflammation and support the proliferation of Newcastle disease virus
出版年份 2019 全文链接
标题
eIF2α-CHOP-BCl-2/JNK and IRE1α-XBP1/JNK signaling promote apoptosis and inflammation and support the proliferation of Newcastle disease virus
作者
关键词
-
出版物
Cell Death & Disease
Volume 10, Issue 12, Pages -
出版商
Springer Science and Business Media LLC
发表日期
2019-11-26
DOI
10.1038/s41419-019-2128-6
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Japanese encephalitis virus activates autophagy through XBP1 and ATF6 ER stress sensors in neuronal cells
- (2017) Manish Sharma et al. JOURNAL OF GENERAL VIROLOGY
- IRE1α Signaling Pathways Involved in Mammalian Cell Fate Determination
- (2016) Jie Wu et al. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY
- Regulation of de novo translation of host cells by manipulation of PERK/PKR and GADD34-PP1 activity during Newcastle disease virus infection
- (2016) Ying Liao et al. JOURNAL OF GENERAL VIROLOGY
- The BCL-2 protein family, BH3-mimetics and cancer therapy
- (2015) A R D Delbridge et al. CELL DEATH AND DIFFERENTIATION
- Oncolytic Newcastle disease virus as a prospective anti-cancer therapy. A biologic agent with potential to break therapy resistance
- (2015) Volker Schirrmacher EXPERT OPINION ON BIOLOGICAL THERAPY
- An interconnected hierarchical model of cell death regulation by the BCL-2 family
- (2015) Hui-Chen Chen et al. NATURE CELL BIOLOGY
- The role of MAPK signalling pathways in the response to endoplasmic reticulum stress
- (2014) Nicola J. Darling et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
- Apoptotic induction of lung adenocarcinoma A549 cells infected by recombinant RVG Newcastle disease virus (rL-RVG) in vitro
- (2014) YULAN YAN et al. Molecular Medicine Reports
- Newcastle disease virus: Current status and our understanding
- (2014) Ketan Ganar et al. VIRUS RESEARCH
- Upregulation of CHOP/GADD153 during Coronavirus Infectious Bronchitis Virus Infection Modulates Apoptosis by Restricting Activation of the Extracellular Signal-Regulated Kinase Pathway
- (2013) Y. Liao et al. JOURNAL OF VIROLOGY
- The unfolded protein response: controlling cell fate decisions under ER stress and beyond
- (2012) Claudio Hetz NATURE REVIEWS MOLECULAR CELL BIOLOGY
- ER Stress Activates NF-κB by Integrating Functions of Basal IKK Activity, IRE1 and PERK
- (2012) Arvin B. Tam et al. PLoS One
- Neurological lesions in chickens experimentally infected with virulent Newcastle disease virus isolates
- (2011) Roselene Ecco et al. AVIAN PATHOLOGY
- Influenza A Viral Replication Is Blocked by Inhibition of the Inositol-requiring Enzyme 1 (IRE1) Stress Pathway
- (2011) Ihab H. Hassan et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Rotavirus Infection Induces the Unfolded Protein Response of the Cell and Controls It through the Nonstructural Protein NSP3
- (2011) V. Trujillo-Alonso et al. JOURNAL OF VIROLOGY
- The Unfolded Protein Response: Integrating Stress Signals Through the Stress Sensor IRE1α
- (2011) Claudio Hetz et al. PHYSIOLOGICAL REVIEWS
- Mitochondria and cell death: outer membrane permeabilization and beyond
- (2010) Stephen W. G. Tait et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- Newcastle Disease Virus Infection Promotes Bax Redistribution to Mitochondria and Cell Death in HeLa Cells
- (2009) Aidin Molouki et al. INTERVIROLOGY
- Regulated Ire1-dependent decay of messenger RNAs in mammalian cells
- (2009) Julie Hollien et al. JOURNAL OF CELL BIOLOGY
- Inhibition of Protein Kinase R Activation and Upregulation of GADD34 Expression Play a Synergistic Role in Facilitating Coronavirus Replication by Maintaining De Novo Protein Synthesis in Virus-Infected Cells
- (2009) X. Wang et al. JOURNAL OF VIROLOGY
- Cell death in the host response to infection
- (2008) K Labbé et al. CELL DEATH AND DIFFERENTIATION
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started