The epithelial Na+ channel α- and γ-subunits are cleaved at predicted furin-cleavage sites, glycosylated and membrane associated in human kidney

Urokinase‐type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)‐mediated sodium retention in experimental nephrotic syndrome

(2019) Bernhard N. Bohnert et al.   Acta Physiologica

Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice

(2018) S. Haerteis et al.   Acta Physiologica

Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome

(2018) Bernhard N. Bohnert et al.   KIDNEY INTERNATIONAL

Pre-eclampsia is associated with altered expression of the renal sodium transporters NKCC2, NCC and ENaC in urinary extracellular vesicles

(2018) Chih-Chiang Hu et al.   PLoS One

Structural assembly of the megadalton-sized receptor for intestinal vitamin B12 uptake and kidney protein reabsorption

(2018) Casper Larsen et al.   Nature Communications

Urine exosomes from healthy and hypertensive pregnancies display elevated level of α-subunit and cleaved α- and γ-subunits of the epithelial sodium channel—ENaC

(2017) Maria R. Nielsen et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

Regulation of ENaC trafficking in rat kidney

(2016) Gustavo Frindt et al.   JOURNAL OF GENERAL PHYSIOLOGY

Activation of the Endogenous Renin-Angiotensin-Aldosterone System or Aldosterone Administration Increases Urinary Exosomal Sodium Channel Excretion

(2015) Y. Qi et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Diabetic nephropathy is associated with increased urine excretion of proteases plasmin, prostasin and urokinase and activation of amiloride-sensitive current in collecting duct cells

(2015) H. Andersen et al.   NEPHROLOGY DIALYSIS TRANSPLANTATION

Review/N-glycans: The making of a varied toolbox

(2015) Nausicaä Lannoo et al.   PLANT SCIENCE

Epithelial Sodium Channel-Mediated Sodium Transport Is Not Dependent on the Membrane-Bound Serine Protease CAP2/Tmprss4

(2015) Anna Keppner et al.   PLoS One

Plasmin in urine from patients with type 2 diabetes and treatment-resistant hypertension activates ENaC in vitro

(2014) Kristian B. Buhl et al.   JOURNAL OF HYPERTENSION

Colon-Specific Deletion of Epithelial Sodium Channel Causes Sodium Loss and Aldosterone Resistance

(2014) S. Malsure et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

The Epithelial Sodium Channel  -Subunit Is Processed Proteolytically in Human Kidney

(2014) R. M. Zachar et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Remission of nephrotic syndrome diminishes urinary plasmin content and abolishes activation of ENaC

(2013) René F. Andersen et al.   PEDIATRIC NEPHROLOGY

Regulation of ion channels by the serum- and glucocorticoid-inducible kinase SGK1

(2012) Florian Lang et al.   FASEB JOURNAL

Prostasin-dependent activation of epithelial Na+ channels by low plasmin concentrations

(2009) Per Svenningsen et al.   AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY

ENaC-mediated alveolar fluid clearance and lung fluid balance depend on the channel-activating protease 1

(2009) Carole Planès et al.   EMBO Molecular Medicine

Activation of the Epithelial Sodium Channel (ENaC) by Serine Proteases

(2008) Bernard C. Rossier et al.   Annual Review of Physiology

Plasmin Activates Epithelial Na+Channels by Cleaving the γ Subunit

(2008) Christopher J. Passero et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Preferential Assembly of Epithelial Sodium Channel (ENaC) Subunits inXenopusOocytes

(2008) Michael Harris et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Plasmin in Nephrotic Urine Activates the Epithelial Sodium Channel

(2008) P. Svenningsen et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY