MicroRNA‐16 is involved in the pathogenesis of pre‐eclampsia via regulation of Notch2
出版年份 2019 全文链接
标题
MicroRNA‐16 is involved in the pathogenesis of pre‐eclampsia via regulation of Notch2
作者
关键词
-
出版物
JOURNAL OF CELLULAR PHYSIOLOGY
Volume -, Issue -, Pages -
出版商
Wiley
发表日期
2019-10-24
DOI
10.1002/jcp.29330
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- The critical role of abnormal trophoblast development in the etiology of preeclampsia
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- Notch2 and Notch3 suppress the proliferation and mediate invasion of trophoblast cell lines
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- Identification of key microRNAs and genes in preeclampsia by bioinformatics analysis
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- MicroRNA-16 inhibits feto-maternal angiogenesis and causes recurrent spontaneous abortion by targeting vascular endothelial growth factor
- (2016) Yongsheng Zhu et al. Scientific Reports
- MiR-132, miR-15a and miR-16 synergistically inhibit pituitary tumor cell proliferation, invasion and migration by targeting Sox5
- (2015) Wang Renjie et al. CANCER LETTERS
- miR-16 targets Bcl-2 in paclitaxel-resistant lung cancer cells and overexpression of miR-16 along with miR-17 causes unprecedented sensitivity by simultaneously modulating autophagy and apoptosis
- (2015) Abhisek Chatterjee et al. CELLULAR SIGNALLING
- Effects of Notch2 and Notch3 on Cell Proliferation and Apoptosis of Trophoblast Cell Lines
- (2015) Wei-Xiu Zhao et al. International Journal of Medical Sciences
- Preeclampsia, Placental Insufficiency, and Autism Spectrum Disorder or Developmental Delay
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- A microRNA-mediated regulatory loop modulates NOTCH and MYC oncogenic signals in B- and T-cell malignancies
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- Short hairpin RNA targeting Notch2 inhibits U87 human glioma cell proliferation by inducing cell cycle arrest and apoptosis in vitro and in vivo
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- Increased LEF1 Expression and Decreased Notch2 Expression Are Strong Predictors of Poor Outcomes in Colorectal Cancer Patients
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