Differential expression of miR‐142‐3p protects cardiomyocytes from myocardial ischemia‐reperfusion via TLR4/NFkB axis
出版年份 2019 全文链接
标题
Differential expression of miR‐142‐3p protects cardiomyocytes from myocardial ischemia‐reperfusion via TLR4/NFkB axis
作者
关键词
-
出版物
JOURNAL OF CELLULAR BIOCHEMISTRY
Volume -, Issue -, Pages -
出版商
Wiley
发表日期
2019-11-20
DOI
10.1002/jcb.29506
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- LncRNA myocardial infarction-associated transcript (MIAT) contributed to cardiac hypertrophy by regulating TLR4 via miR-93
- (2018) Yunwei Li et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- Downregulation of TLR4 by miR-181a Provides Negative Feedback Regulation to Lipopolysaccharide-Induced Inflammation
- (2018) Kangfeng Jiang et al. Frontiers in Pharmacology
- Exosome-encapsulated miR-6089 regulates inflammatory response via targeting TLR4
- (2018) Donghua Xu et al. JOURNAL OF CELLULAR PHYSIOLOGY
- Baicalin alleviates H2O2-induced injury of H9c2 cardiomyocytes through suppression of the Wnt/β-catenin signaling pathway
- (2017) Liman Qiu et al. Molecular Medicine Reports
- The role of Toll-like receptor 4 (TLR4) in cardiac ischaemic-reperfusion injury, cardioprotection and preconditioning
- (2016) Sam Man Lee et al. CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY
- BDNF-mediates Down-regulation of MicroRNA-195 Inhibits Ischemic Cardiac Apoptosis in Rats
- (2016) Pengzhou Hang et al. International Journal of Biological Sciences
- Radioprotective 105 kDa protein attenuates ischemia/reperfusion-induced myocardial apoptosis and autophagy by inhibiting the activation of the TLR4/NF-κB signaling pathway in rats
- (2016) Xin Guo et al. INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE
- MicroRNA-93 inhibits ischemia-reperfusion induced cardiomyocyte apoptosis by targeting PTEN
- (2016) Zun-Ping Ke et al. Oncotarget
- Combination of microRNA-21 and microRNA-146a Attenuates Cardiac Dysfunction and Apoptosis During Acute Myocardial Infarction in Mice
- (2016) Wei Huang et al. Molecular Therapy-Nucleic Acids
- miR-98 protects endothelial cells against hypoxia/reoxygenation induced-apoptosis by targeting caspase-3
- (2015) He-wen Li et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- RP105 Protects Against Apoptosis in Ischemia/Reperfusion-Induced Myocardial Damage in Rats by Suppressing TLR4-Mediated Signaling Pathways
- (2015) Jun Yang et al. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY
- miR-378b Promotes Differentiation of Keratinocytes through NKX3.1
- (2015) Xi-liang Wang et al. PLoS One
- NFkappaB activation is essential for miR-21 induction by TGFβ1 in high glucose conditions
- (2014) Radha Madhyastha et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Cardioprotective effect of carvedilol: inhibition of apoptosis in H9c2 cardiomyocytes via the TLR4/NF-κB pathway following ischemia/reperfusion injury
- (2014) YONG ZHAO et al. Experimental and Therapeutic Medicine
- Inhibition of MicroRNA-92a Protects Against Ischemia/Reperfusion Injury in a Large-Animal Model
- (2013) Rabea Hinkel et al. CIRCULATION
- The HMGB1–TLR4 axis contributes to myocardial ischemia/reperfusion injury via regulation of cardiomyocyte apoptosis
- (2013) Hua-Sheng Ding et al. GENE
- MiR-34a is involved in Tat-induced HIV-1 long terminal repeat (LTR) transactivation through the SIRT1/NFκB pathway
- (2012) Hong-Sheng Zhang et al. FEBS LETTERS
- Ischemic Postconditioning Mediates Cardioprotection Via PI3K/GSK-3β/β-Catenin Signaling Pathway in Ischemic Rat Myocardium
- (2012) Qiao-ling Wu et al. SHOCK
- In search of new therapeutic targets and strategies for heart failure: recent advances in basic science
- (2011) Ajay M Shah et al. LANCET
- MicroRNA-320 Is Involved in the Regulation of Cardiac Ischemia/Reperfusion Injury by Targeting Heat-Shock Protein 20
- (2009) Xiao-Ping Ren et al. CIRCULATION
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