期刊
JOURNAL OF INVESTIGATIVE DERMATOLOGY
卷 139, 期 5, 页码 995-996出版社
ELSEVIER SCIENCE INC
DOI: 10.1016/j.jid.2019.01.026
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Psoriasis is a T helper type 17-mediated immune disease. Initial triggers that lead to T helper type 17 production and inflammatory cell recruitment into skin are being delineated. Autoantigens that stimulate T helper type 17 cells are also being identified. A new and important piece of the puzzle indicates that keratinocytes not only amplify inflammation, but that they are essential for a full-blown IL-17-mediated psoriatic phenotype in mice.
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